Dimethyl phthalate


Curated chemical-gene interactions from CTD
Entrez gene IDGene symbolInteraction typeReference
10257 ABCC4 Increases expression PMID:35739735
1244 ABCC2 Increases expression PMID:35739735
1544 CYP1A2 Decreases activity PMID:27432241
1583 CYP11A1 Increases expression PMID:26472102
24300 CYP2B1 Decreases activity PMID:27432241
3458 IFNG Decreases expression PMID:35598668
3479 IGF1 Decreases expression PMID:26472102
355 FAS Increases expression PMID:35598668
356 FASLG Increases expression PMID:35598668
3565 IL4 Decreases expression PMID:35598668
3569 IL6 Decreases expression PMID:35598668
4436 MSH2 Decreases expression PMID:26472102
578 BAK1 Increases expression PMID:35598668
581 BAX Increases expression PMID:35598668
596 BCL2 Increases expression PMID:35598668
637 BID Increases expression PMID:35598668
6647 SOD1 Decreases expression PMID:35598668
6770 STAR Decreases expression PMID:26472102
835 CASP2 Increases expression PMID:35598668
836 CASP3 Increases expression PMID:35598668
840 CASP7 Increases expression PMID:35598668
841 CASP8 Increases expression PMID:35598668
8714 ABCC3 Increases expression PMID:35739735
8772 FADD Increases expression PMID:35598668
93649 MYOCD Decreases expression PMID:26472102
9429 ABCG2 Increases expression PMID:35739735
949 SCARB1 Decreases expression PMID:26472102

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.