p-Nitrophenol


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000084 Mitotic s phase Decreases phenotype PMID:34673133
GO:0000086 G2/m transition of mitotic cell cycle Affects phenotype PMID:34673133
GO:0006915 Apoptotic process Increases phenotype PMID:34673133
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:26739637
GO:0007005 Mitochondrion organization Decreases phenotype PMID:34673133
GO:0008209 Androgen metabolic process Affects phenotype PMID:24070737
GO:0008210 Estrogen metabolic process Affects phenotype PMID:24070737
GO:0008283 Cell population proliferation Decreases phenotype PMID:34673133
GO:0009790 Embryo development Decreases phenotype PMID:39510238
GO:0016712 Oxidoreductase activity, acting on paired donors, with incorporation or reduction of molecular oxygen, reduced flavin or flavoprotein as one donor, and incorporation of one atom of oxygen Decreases phenotype PMID:32805337
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:34673133
GO:0034440 Lipid oxidation Increases phenotype PMID:23997041
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:23997041
GO:0044237 Cellular metabolic process Decreases phenotype PMID:34673133
GO:0045023 G0 to g1 transition Increases phenotype PMID:34673133
GO:0048640 Negative regulation of developmental growth Increases phenotype PMID:39510238
GO:0070365 Hepatocyte differentiation Increases phenotype PMID:26739637
GO:0070995 Nadph oxidation Increases phenotype PMID:22499231
GO:0097189 Apoptotic body Increases phenotype PMID:34673133
GO:0140042 Lipid droplet formation Increases phenotype PMID:34673133
GO:1901671 Positive regulation of superoxide dismutase activity Decreases phenotype PMID:23997041
GO:1903047 Mitotic cell cycle process Increases phenotype PMID:34673133
GO:1903284 Positive regulation of glutathione peroxidase activity Decreases phenotype PMID:23997041
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:23997041
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.