| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0000723 | Telomere maintenance | Decreases phenotype | PMID:19772576 |
| GO:0001508 | Action potential | Affects phenotype | PMID:32234516 |
| GO:0001776 | Leukocyte homeostasis | Affects phenotype | PMID:31988427 |
| GO:0005245 | Voltage-gated calcium channel activity | Decreases phenotype | PMID:29925288 |
| GO:0005248 | Voltage-gated sodium channel activity | Affects phenotype | PMID:29925288 |
| GO:0006750 | Glutathione biosynthetic process | Decreases phenotype | PMID:24733833 |
| GO:0006809 | Nitric oxide biosynthetic process | Increases phenotype | PMID:24733833 |
| GO:0007088 | Regulation of mitotic nuclear division | Decreases phenotype | PMID:23138419 |
| GO:0007271 | Synaptic transmission, cholinergic | Affects phenotype | PMID:30097702 |
| GO:0007613 | Memory | Decreases phenotype | PMID:29935984; PMID:29935984; PMID:32980479; PMID:33086094; PMID:32234516; PMID:32980479; PMID:33086094 |
| GO:0008344 | Adult locomotory behavior | Affects phenotype | PMID:29935984 |
| GO:0010424 | Dna methylation on cytosine within a cg sequence | Affects phenotype | PMID:33438815 |
| GO:0010460 | Positive regulation of heart rate | Increases phenotype | PMID:29925288 |
| GO:0023041 | Neuronal signal transduction | Affects phenotype | PMID:32980479 |
| GO:0030097 | Hemopoiesis | Affects phenotype | PMID:31988427 |
| GO:0030220 | Platelet formation | Affects phenotype | PMID:31988427 |
| GO:0035176 | Social behavior | Decreases phenotype | PMID:29935984; PMID:29935984; PMID:33086094; PMID:32980479; PMID:33086094 |
| GO:0035249 | Synaptic transmission, glutamatergic | Affects phenotype | PMID:30097702 |
| GO:0035934 | Corticosterone secretion | Increases phenotype | PMID:37944760 |
| GO:0042403 | Thyroid hormone metabolic process | Increases phenotype | PMID:37944760 |
| GO:0048167 | Regulation of synaptic plasticity | Affects phenotype | PMID:30097702 |
| GO:0050879 | Multicellular organismal movement | Increases phenotype | PMID:32980479 |
| GO:0060291 | Long-term synaptic potentiation | Decreases phenotype | PMID:33086094 |
| GO:0061900 | Glial cell activation | Increases phenotype | PMID:32234516 |
| GO:0090648 | Response to environmental enrichment | Decreases phenotype | PMID:33086094 |
| GO:0098989 | Nmda selective glutamate receptor signaling pathway | Affects phenotype | PMID:30097702 |
| GO:0099565 | Chemical synaptic transmission, postsynaptic | Affects phenotype | PMID:33086094 |
| GO:1900996 | Benzene catabolic process | Decreases phenotype | PMID:30086327 |
| GO:2000310 | Regulation of nmda receptor activity | Affects phenotype | PMID:30097702 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.