2,4,6-Tribromophenol


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0004457 Lactate dehydrogenase activity Increases phenotype PMID:14599456
GO:0006915 Apoptotic process Increases phenotype PMID:14599456
GO:0008219 Cell death Increases phenotype PMID:29427708
GO:0008283 Cell population proliferation Decreases phenotype PMID:14599456; PMID:29427708
GO:0022008 Neurogenesis Increases phenotype PMID:14599456
GO:0030154 Cell differentiation Increases phenotype PMID:14599456
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:14599456
GO:0042910 Xenobiotic transmembrane transporter activity Decreases phenotype PMID:29427708
GO:0044237 Cellular metabolic process Increases phenotype PMID:14599456
GO:0044319 Wound healing, spreading of cells Decreases phenotype PMID:29427708
GO:0051348 Negative regulation of transferase activity Affects phenotype PMID:29331935
GO:0051923 Sulfation Decreases phenotype PMID:29331935
GO:0080009 Mrna methylation Increases phenotype PMID:38157273
GO:0097189 Apoptotic body Increases phenotype PMID:14599456
GO:0097484 Dendrite extension Increases phenotype PMID:14599456
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.