Acephate


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0002021 Response to dietary excess Affects phenotype PMID:31326399
GO:0004364 Glutathione transferase activity Increases phenotype PMID:33217513
GO:0004602 Glutathione peroxidase activity Increases phenotype PMID:33217513
GO:0006094 Gluconeogenesis Increases phenotype PMID:22778074
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:33217513
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:29149346
GO:0016042 Lipid catabolic process Affects phenotype PMID:33217513
GO:0019432 Triglyceride biosynthetic process Increases phenotype PMID:22778074
GO:0031959 Mineralocorticoid receptor signaling pathway Increases phenotype PMID:29149346
GO:0035774 Positive regulation of insulin secretion involved in cellular response to glucose stimulus Affects phenotype PMID:31326399
GO:0040018 Positive regulation of multicellular organism growth Affects phenotype PMID:31326399
GO:0042593 Glucose homeostasis Decreases phenotype PMID:31326399
GO:0048729 Tissue morphogenesis Increases phenotype PMID:31326399
GO:0050890 Cognition Decreases phenotype PMID:27453326
GO:0061179 Negative regulation of insulin secretion involved in cellular response to glucose stimulus Decreases phenotype PMID:31326399
GO:1904178 Negative regulation of adipose tissue development Decreases phenotype PMID:31326399
GO:1904179 Positive regulation of adipose tissue development Increases phenotype PMID:31326399
GO:2000078 Positive regulation of type b pancreatic cell development Increases phenotype PMID:31326399
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.