Simazine


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0006805 Xenobiotic metabolic process Decreases phenotype PMID:30545405
GO:0006915 Apoptotic process Increases phenotype PMID:25461544
GO:0008283 Cell population proliferation Affects phenotype PMID:25461544
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:29137186; PMID:32822773
GO:0015872 Dopamine transport Affects phenotype PMID:29115391
GO:0030521 Androgen receptor signaling pathway Increases phenotype PMID:19368227
GO:0033484 Cellular nitric oxide homeostasis Affects phenotype PMID:27431321
GO:0035641 Locomotory exploration behavior Decreases phenotype PMID:34936186
GO:0036269 Swimming behavior Decreases phenotype PMID:34936186
GO:0042053 Regulation of dopamine metabolic process Increases phenotype PMID:29137186
GO:0042416 Dopamine biosynthetic process Decreases phenotype PMID:29115391
GO:0042417 Dopamine metabolic process Increases phenotype PMID:32822773; PMID:34936186
GO:1904480 Positive regulation of intestinal absorption Increases phenotype PMID:30545405

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.