7,12-Dimethylbenz(a)anthracene


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000902 Cell morphogenesis Affects phenotype PMID:37840363
GO:0001836 Release of cytochrome c from mitochondria Increases phenotype PMID:35908932
GO:0002639 Positive regulation of immunoglobulin production Increases phenotype PMID:27869817
GO:0003958 Nadph-hemoprotein reductase activity Increases phenotype PMID:27091720
GO:0004035 Alkaline phosphatase activity Affects phenotype PMID:16025432; PMID:33063951
GO:0004069 L-aspartate:2-oxoglutarate aminotransferase activity Affects phenotype PMID:32623939; PMID:33063951; PMID:39214269
GO:0004111 Creatine kinase activity Increases phenotype PMID:39214269
GO:0004364 Glutathione transferase activity Increases phenotype PMID:18678235; PMID:21801490; PMID:23033690; PMID:27091720; PMID:31476248; PMID:33063951; PMID:35243731
GO:0004457 Lactate dehydrogenase activity Affects phenotype PMID:32623939; PMID:33063951; PMID:39214269
GO:0004602 Glutathione peroxidase activity Affects phenotype PMID:24995432; PMID:25744308; PMID:31476248; PMID:31476248; PMID:33759307; PMID:33063951; PMID:33347680; PMID:33347680; PMID:35243731; PMID:37840363; PMID:33347680; PMID:37840363; PMID:33759307; PMID:35243731; PMID:37840363
GO:0004784 Superoxide dismutase activity Decreases phenotype PMID:24995432; PMID:25744308; PMID:31476248; PMID:31476248; PMID:33759307; PMID:32662567; PMID:33063951; PMID:33347680; PMID:33347680; PMID:35243731; PMID:37840363; PMID:33347680; PMID:37840363; PMID:33759307; PMID:35243731; PMID:37840363
GO:0005243 Gap junction channel activity Decreases phenotype PMID:36863560
GO:0006309 Apoptotic dna fragmentation Increases phenotype PMID:24995432
GO:0006417 Regulation of translation Affects phenotype PMID:39910959
GO:0006606 Protein import into nucleus Affects phenotype PMID:33347680
GO:0006749 Glutathione metabolic process Increases phenotype PMID:18678235; PMID:18678235; PMID:32623939; PMID:33063951; PMID:21801490; PMID:21801490; PMID:33347680; PMID:35243731; PMID:37840363; PMID:21801490; PMID:35243731; PMID:37840363; PMID:23033690; PMID:31476248; PMID:31476248; PMID:33759307; PMID:32623939; PMID:32623939; PMID:33063951; PMID:32662567; PMID:33063951; PMID:33347680; PMID:33347680; PMID:37840363; PMID:33759307; PMID:34896434; PMID:35243731; PMID:37840363
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:24995432; PMID:25744308
GO:0006809 Nitric oxide biosynthetic process Increases phenotype PMID:16025432; PMID:24995432
GO:0006915 Apoptotic process Decreases phenotype PMID:31332895; PMID:34896434
GO:0006954 Inflammatory response Increases phenotype PMID:23835587; PMID:39214269
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:23717041; PMID:25448685
GO:0008219 Cell death Increases phenotype PMID:12734332; PMID:35908932
GO:0008283 Cell population proliferation Increases phenotype PMID:27091720; PMID:29663660; PMID:37352108; PMID:38299738; PMID:39214269
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:23835587; PMID:26945725
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:29555536
GO:0008360 Regulation of cell shape Affects phenotype PMID:29663660
GO:0008610 Lipid biosynthetic process Increases phenotype PMID:30027798
GO:0009636 Response to toxic substance Affects phenotype PMID:35364107
GO:0010820 Positive regulation of t cell chemotaxis Increases phenotype PMID:27869817
GO:0015020 Glucuronosyltransferase activity Decreases phenotype PMID:27091720
GO:0016042 Lipid catabolic process Increases phenotype PMID:25744308; PMID:31476248; PMID:32662567; PMID:33063951; PMID:33347680; PMID:33347680; PMID:35243731; PMID:33347706; PMID:33759307; PMID:35243731
GO:0016887 Atp hydrolysis activity Decreases phenotype PMID:27091720
GO:0018158 Protein oxidation Increases phenotype PMID:27091720; PMID:32623939
GO:0019319 Hexose biosynthetic process Increases phenotype PMID:27091720
GO:0019627 Urea metabolic process Affects phenotype PMID:32623939
GO:0019825 Oxygen binding Increases phenotype PMID:27091720
GO:0019852 L-ascorbic acid metabolic process Affects phenotype PMID:31476248; PMID:31476248; PMID:33759307; PMID:32662567; PMID:33063951; PMID:33347680; PMID:33347680; PMID:35243731; PMID:33347680; PMID:35243731; PMID:37840363; PMID:33759307; PMID:35243731; PMID:37840363
GO:0030154 Cell differentiation Increases phenotype PMID:38299738
GO:0030199 Collagen fibril organization Increases phenotype PMID:39214269
GO:0034440 Lipid oxidation Increases phenotype PMID:21801490; PMID:21801490; PMID:37840363; PMID:23033690; PMID:32623939; PMID:37840363
GO:0040015 Negative regulation of multicellular organism growth Increases phenotype PMID:23033690
GO:0042360 Vitamin e metabolic process Affects phenotype PMID:21801490; PMID:21801490; PMID:33347680; PMID:35243731; PMID:37840363; PMID:31476248; PMID:31476248; PMID:33759307; PMID:32662567; PMID:33347680; PMID:33347680; PMID:37840363; PMID:33759307; PMID:35243731; PMID:37840363
GO:0042554 Superoxide anion generation Increases phenotype PMID:24995432
GO:0043616 Keratinocyte proliferation Decreases phenotype PMID:38299738
GO:0044242 Cellular lipid catabolic process Increases phenotype PMID:24995432
GO:0044319 Wound healing, spreading of cells Increases phenotype PMID:29663660
GO:0045454 Cell redox homeostasis Affects phenotype PMID:30027798
GO:0045618 Positive regulation of keratinocyte differentiation Increases phenotype PMID:27869817
GO:0046209 Nitric oxide metabolic process Affects phenotype PMID:32623939; PMID:33063951; PMID:33063951
GO:0046415 Urate metabolic process Affects phenotype PMID:32623939
GO:0046449 Creatinine metabolic process Affects phenotype PMID:32623939
GO:0046466 Membrane lipid catabolic process Increases phenotype PMID:30027798; PMID:34972512; PMID:6413056
GO:0046621 Negative regulation of organ growth Increases phenotype PMID:23033690
GO:0046622 Positive regulation of organ growth Increases phenotype PMID:29877212
GO:0046827 Positive regulation of protein export from nucleus Increases phenotype PMID:21294050
GO:0047823 D-glutamyltransferase activity Affects phenotype PMID:33063951
GO:0051347 Positive regulation of transferase activity Increases phenotype PMID:21801490
GO:0051348 Negative regulation of transferase activity Affects phenotype PMID:21801490; PMID:23033690
GO:0051882 Mitochondrial depolarization Increases phenotype PMID:35908932
GO:0060384 Innervation Increases phenotype PMID:27869817
GO:0070265 Necrotic cell death Increases phenotype PMID:39214269
GO:0070994 Detection of oxidative stress Increases phenotype PMID:39214269
GO:0072089 Stem cell proliferation Decreases phenotype PMID:38299738
GO:0072593 Reactive oxygen species metabolic process Increases phenotype PMID:25697376; PMID:35908932
GO:0090200 Positive regulation of release of cytochrome c from mitochondria Decreases phenotype PMID:25478867
GO:0097237 Cellular response to toxic substance Increases phenotype PMID:29502735
GO:0097531 Mast cell migration Increases phenotype PMID:37352108
GO:0098754 Detoxification Increases phenotype PMID:21801490
GO:1901670 Negative regulation of superoxide dismutase activity Increases phenotype PMID:21801490; PMID:23033690; PMID:34972512
GO:1902034 Negative regulation of hematopoietic stem cell proliferation Increases phenotype PMID:32553695
GO:1903283 Negative regulation of glutathione peroxidase activity Increases phenotype PMID:21801490; PMID:23033690
GO:1903284 Positive regulation of glutathione peroxidase activity Increases phenotype PMID:21801490
GO:1903604 Cytochrome metabolic process Affects phenotype PMID:37840363
GO:1903605 Cytochrome biosynthetic process Increases phenotype PMID:30027798
GO:2000354 Regulation of ovarian follicle development Affects phenotype PMID:24576726
GO:2001244 Positive regulation of intrinsic apoptotic signaling pathway Decreases phenotype PMID:25478867

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.