Etomidate


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0006123 Mitochondrial electron transport, cytochrome c to oxygen Decreases phenotype PMID:25421967
GO:0006650 Glycerophospholipid metabolic process Decreases phenotype PMID:38604439
GO:0006749 Glutathione metabolic process Affects phenotype PMID:32679257
GO:0009449 Gamma-aminobutyric acid biosynthetic process Decreases phenotype PMID:38604439
GO:0010918 Positive regulation of mitochondrial membrane potential Decreases phenotype PMID:25421967
GO:0014053 Negative regulation of gamma-aminobutyric acid secretion Increases phenotype PMID:38604439
GO:0014063 Negative regulation of serotonin secretion Increases phenotype PMID:38604439
GO:0018158 Protein oxidation Affects phenotype PMID:32679257
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:25421967; PMID:38604439
GO:0044237 Cellular metabolic process Affects phenotype PMID:32679257
GO:0072660 Maintenance of protein location in plasma membrane Affects phenotype PMID:29320738
GO:0090200 Positive regulation of release of cytochrome c from mitochondria Increases phenotype PMID:25421967
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:25421967
GO:1905628 Negative regulation of serotonin biosynthetic process Increases phenotype PMID:38604439
GO:2001024 Negative regulation of response to drug Increases phenotype PMID:38604439

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.