| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0002260 | Lymphocyte homeostasis | Affects phenotype | PMID:25673971 |
| GO:0006590 | Thyroid hormone generation | Decreases phenotype | PMID:30991129 |
| GO:0006750 | Glutathione biosynthetic process | Decreases phenotype | PMID:30991129 |
| GO:0006915 | Apoptotic process | Increases phenotype | PMID:11834455 |
| GO:0006974 | Cellular response to dna damage stimulus | Increases phenotype | PMID:30991129 |
| GO:0016042 | Lipid catabolic process | Increases phenotype | PMID:30991129 |
| GO:0034101 | Erythrocyte homeostasis | Affects phenotype | PMID:25673971 |
| GO:0046879 | Hormone secretion | Increases phenotype | PMID:20439182 |
| GO:0048589 | Developmental growth | Decreases phenotype | PMID:26151950 |
| GO:0055072 | Iron ion homeostasis | Affects phenotype | PMID:25673971 |
| GO:0070460 | Thyroid-stimulating hormone secretion | Increases phenotype | PMID:20439182; PMID:26408806 |
| GO:2000609 | Regulation of thyroid hormone generation | Affects phenotype | PMID:26485730 |
| GO:2000610 | Negative regulation of thyroid hormone generation | Increases phenotype | PMID:35385113 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.