| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0006006 | Glucose metabolic process | Affects phenotype | PMID:32745781 |
| GO:0006083 | Acetate metabolic process | Affects phenotype | PMID:32745781 |
| GO:0006105 | Succinate metabolic process | Affects phenotype | PMID:32745781 |
| GO:0006631 | Fatty acid metabolic process | Affects phenotype | PMID:32745781 |
| GO:0006749 | Glutathione metabolic process | Affects phenotype | PMID:28364781 |
| GO:0008203 | Cholesterol metabolic process | Affects phenotype | PMID:32745781 |
| GO:0008283 | Cell population proliferation | Increases phenotype | PMID:32736067 |
| GO:0010729 | Positive regulation of hydrogen peroxide biosynthetic process | Increases phenotype | PMID:28364781 |
| GO:0018158 | Protein oxidation | Increases phenotype | PMID:28364781 |
| GO:0019627 | Urea metabolic process | Affects phenotype | PMID:32745781 |
| GO:0019852 | L-ascorbic acid metabolic process | Affects phenotype | PMID:28364781 |
| GO:0031987 | Locomotion involved in locomotory behavior | Decreases phenotype | PMID:32736067 |
| GO:0034440 | Lipid oxidation | Increases phenotype | PMID:28364781 |
| GO:0035176 | Social behavior | Affects phenotype | PMID:32736067 |
| GO:0050872 | White fat cell differentiation | Increases phenotype | PMID:32745781 |
| GO:1901671 | Positive regulation of superoxide dismutase activity | Affects phenotype | PMID:28364781 |
| GO:1903284 | Positive regulation of glutathione peroxidase activity | Affects phenotype | PMID:28364781 |
| GO:1903428 | Positive regulation of reactive oxygen species biosynthetic process | Increases phenotype | PMID:35395341 |
| GO:1903626 | Positive regulation of dna catabolic process | Increases phenotype | PMID:28364781 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.