| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0006641 | Triglyceride metabolic process | Affects phenotype | PMID:32810590 |
| GO:0006695 | Cholesterol biosynthetic process | Increases phenotype | PMID:28454766 |
| GO:0006703 | Estrogen biosynthetic process | Decreases phenotype | PMID:20132345 |
| GO:0007631 | Feeding behavior | Affects phenotype | PMID:35436446 |
| GO:0008219 | Cell death | Increases phenotype | PMID:29294346 |
| GO:0042756 | Drinking behavior | Affects phenotype | PMID:35436446 |
| GO:0045795 | Positive regulation of cell volume | Increases phenotype | PMID:29038839 |
| GO:0047823 | D-glutamyltransferase activity | Increases phenotype | PMID:29038839 |
| GO:0050810 | Regulation of steroid biosynthetic process | Affects phenotype | PMID:26781511 |
| GO:0061370 | Testosterone biosynthetic process | Decreases phenotype | PMID:20132345 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.