3-Methylquercetin


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001516 Prostaglandin biosynthetic process Increases phenotype PMID:21341175
GO:0002675 Positive regulation of acute inflammatory response Affects phenotype PMID:23774260
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:27151496
GO:0006915 Apoptotic process Increases phenotype PMID:16014620; PMID:22992727
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:17045724
GO:0006979 Response to oxidative stress Affects phenotype PMID:24211276
GO:0007252 I-kappab phosphorylation Increases phenotype PMID:23774260
GO:0008219 Cell death Increases phenotype PMID:25203460
GO:0008283 Cell population proliferation Decreases phenotype PMID:22992727; PMID:25203460
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:37537191
GO:0010942 Positive regulation of cell death Increases phenotype PMID:19222219; PMID:24211276; PMID:24337631
GO:0016042 Lipid catabolic process Increases phenotype PMID:27151496
GO:0016887 Atp hydrolysis activity Increases phenotype PMID:34217736
GO:0032308 Positive regulation of prostaglandin secretion Increases phenotype PMID:24337631
GO:0032964 Collagen biosynthetic process Increases phenotype PMID:27151496
GO:0042307 Positive regulation of protein import into nucleus Increases phenotype PMID:24337631
GO:0042310 Vasoconstriction Increases phenotype PMID:17374653; PMID:18436224
GO:0042311 Vasodilation Increases phenotype PMID:18436224
GO:0042438 Melanin biosynthetic process Increases phenotype PMID:24853321
GO:0042554 Superoxide anion generation Increases phenotype PMID:17374653; PMID:18436224; PMID:25562607
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:37537191
GO:0043123 Positive regulation of i-kappab kinase/nf-kappab signaling Affects phenotype PMID:23774260
GO:0043507 Positive regulation of jun kinase activity Affects phenotype PMID:23774260
GO:0045429 Positive regulation of nitric oxide biosynthetic process Affects phenotype PMID:23774260
GO:0045930 Negative regulation of mitotic cell cycle Increases phenotype PMID:22992727; PMID:37537191
GO:0051000 Positive regulation of nitric-oxide synthase activity Affects phenotype PMID:23774260
GO:0051092 Positive regulation of nf-kappab transcription factor activity Affects phenotype PMID:23774260
GO:0051897 Positive regulation of protein kinase b signaling Affects phenotype PMID:23774260
GO:1901224 Positive regulation of nik/nf-kappab signaling Affects phenotype PMID:23774260
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:17045724; PMID:19222219; PMID:26887929; PMID:27151496
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:24337631
GO:2000377 Regulation of reactive oxygen species metabolic process Affects phenotype PMID:24211276
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.