| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0001776 | Leukocyte homeostasis | Affects phenotype | PMID:31988427 |
| GO:0006749 | Glutathione metabolic process | Decreases phenotype | PMID:21241565 |
| GO:0007088 | Regulation of mitotic nuclear division | Decreases phenotype | PMID:23138419 |
| GO:0010424 | Dna methylation on cytosine within a cg sequence | Affects phenotype | PMID:33438815 |
| GO:0030097 | Hemopoiesis | Affects phenotype | PMID:31988427 |
| GO:0030220 | Platelet formation | Affects phenotype | PMID:31988427 |
| GO:0032286 | Central nervous system myelin maintenance | Decreases phenotype | PMID:21241565 |
| GO:0034440 | Lipid oxidation | Increases phenotype | PMID:21241565; PMID:25518993 |
| GO:0043065 | Positive regulation of apoptotic process | Increases phenotype | PMID:21241565; PMID:25518993 |
| GO:0055075 | Potassium ion homeostasis | Affects phenotype | PMID:33722544 |
| GO:1901671 | Positive regulation of superoxide dismutase activity | Decreases phenotype | PMID:24053957; PMID:25518993 |
| GO:1903284 | Positive regulation of glutathione peroxidase activity | Decreases phenotype | PMID:24053957; PMID:25518993 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.