Styrene


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001776 Leukocyte homeostasis Affects phenotype PMID:31988427
GO:0002260 Lymphocyte homeostasis Affects phenotype PMID:36912746
GO:0004346 Glucose-6-phosphatase activity Increases phenotype PMID:28678435
GO:0004611 Phosphoenolpyruvate carboxykinase activity Decreases phenotype PMID:28678435
GO:0004784 Superoxide dismutase activity Increases phenotype PMID:28678435
GO:0006813 Potassium ion transport Affects phenotype PMID:32540163
GO:0006997 Nucleus organization Affects phenotype PMID:33722544
GO:0007033 Vacuole organization Increases phenotype PMID:33722544
GO:0008184 Glycogen phosphorylase activity Decreases phenotype PMID:28678435
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:28962520
GO:0016042 Lipid catabolic process Increases phenotype PMID:28678435
GO:0018158 Protein oxidation Increases phenotype PMID:28678435
GO:0030097 Hemopoiesis Affects phenotype PMID:31988427
GO:0043576 Regulation of respiratory gaseous exchange Affects phenotype PMID:25108490
GO:0055075 Potassium ion homeostasis Affects phenotype PMID:33722544
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:28678435
GO:2000401 Regulation of lymphocyte migration Affects phenotype PMID:36912746
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.