4-Nitro-3-cresol


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001541 Ovarian follicle development Affects phenotype PMID:35654127
GO:0004602 Glutathione peroxidase activity Decreases phenotype PMID:22467373
GO:0004784 Superoxide dismutase activity Decreases phenotype PMID:22467373
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:22467373
GO:0006915 Apoptotic process Increases phenotype PMID:22467373
GO:0007286 Spermatid development Decreases phenotype PMID:29292090
GO:0009395 Phospholipid catabolic process Decreases phenotype PMID:29292090
GO:0009790 Embryo development Decreases phenotype PMID:39510238
GO:0010669 Epithelial structure maintenance Decreases phenotype PMID:29307546
GO:0016042 Lipid catabolic process Increases phenotype PMID:22467373
GO:0035710 Cd4-positive, alpha-beta t cell activation Increases phenotype PMID:35910793
GO:0035712 T-helper 2 cell activation Increases phenotype PMID:35910793
GO:0035745 T-helper 2 cell cytokine production Increases phenotype PMID:35910793
GO:0042110 T cell activation Increases phenotype PMID:35910793
GO:0045590 Negative regulation of regulatory t cell differentiation Increases phenotype PMID:36283488
GO:0048640 Negative regulation of developmental growth Increases phenotype PMID:39510238
GO:0050665 Hydrogen peroxide biosynthetic process Increases phenotype PMID:22467373
GO:1900138 Negative regulation of phospholipase a2 activity Increases phenotype PMID:29292090
GO:1901318 Negative regulation of flagellated sperm motility Increases phenotype PMID:29292090
GO:1904037 Positive regulation of epithelial cell apoptotic process Increases phenotype PMID:29307546
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.