Dibromoacetic acid


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:29155130
GO:0010918 Positive regulation of mitochondrial membrane potential Decreases phenotype PMID:29155130
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:29155130
GO:0046330 Positive regulation of jnk cascade Increases phenotype PMID:29155130
GO:0070374 Positive regulation of erk1 and erk2 cascade Increases phenotype PMID:29155130
GO:1900745 Positive regulation of p38mapk cascade Increases phenotype PMID:29155130

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.