Benomyl


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000278 Mitotic cell cycle Decreases phenotype PMID:19049291
GO:0004030 Aldehyde dehydrogenase [nad(p)+] activity Decreases phenotype PMID:24491970
GO:0006874 Cellular calcium ion homeostasis Affects phenotype PMID:27784618
GO:0006915 Apoptotic process Increases phenotype PMID:33930522
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:19591892; PMID:33930522
GO:0008283 Cell population proliferation Decreases phenotype PMID:19049291
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:25324206
GO:0010942 Positive regulation of cell death Increases phenotype PMID:20654549
GO:0010972 Negative regulation of g2/m transition of mitotic cell cycle Increases phenotype PMID:25543211
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:25530041
GO:0043966 Histone h3 acetylation Increases phenotype PMID:25543211
GO:0044237 Cellular metabolic process Decreases phenotype PMID:33930522
GO:0045931 Positive regulation of mitotic cell cycle Decreases phenotype PMID:25530041
GO:0060765 Regulation of androgen receptor signaling pathway Affects phenotype PMID:25324206
GO:0070507 Regulation of microtubule cytoskeleton organization Decreases phenotype PMID:25543211
GO:0071629 Cytoplasm protein quality control by the ubiquitin-proteasome system Decreases phenotype PMID:23988235
GO:0071929 Alpha-tubulin acetylation Increases phenotype PMID:25543211
GO:0072593 Reactive oxygen species metabolic process Affects phenotype PMID:33930522
GO:1901165 Positive regulation of trophoblast cell migration Decreases phenotype PMID:25530041
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:27784618
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.