Tributyl phosphate


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001958 Endochondral ossification Decreases phenotype PMID:33367866
GO:0004364 Glutathione transferase activity Increases phenotype PMID:30974155
GO:0004457 Lactate dehydrogenase activity Increases phenotype PMID:32662595
GO:0008283 Cell population proliferation Increases phenotype PMID:26701863
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:26979758
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:30974155
GO:0009060 Aerobic respiration Increases phenotype PMID:30966736
GO:0033148 Positive regulation of intracellular estrogen receptor signaling pathway Increases phenotype PMID:26979758
GO:0042632 Cholesterol homeostasis Decreases phenotype PMID:30966736
GO:0044237 Cellular metabolic process Decreases phenotype PMID:32662595
GO:0046034 Atp metabolic process Affects phenotype PMID:30966736
GO:0051347 Positive regulation of transferase activity Increases phenotype PMID:30974155
GO:0070050 Neuron cellular homeostasis Decreases phenotype PMID:37120036
GO:0070265 Necrotic cell death Increases phenotype PMID:32662595
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:38603619
GO:2000866 Positive regulation of estradiol secretion Increases phenotype PMID:38603619
GO:2000872 Positive regulation of progesterone secretion Increases phenotype PMID:38603619
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.