Prochloraz


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0006641 Triglyceride metabolic process Affects phenotype PMID:32810590; PMID:34273450; PMID:34273450
GO:0006695 Cholesterol biosynthetic process Increases phenotype PMID:28454766
GO:0008210 Estrogen metabolic process Affects phenotype PMID:37551828
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:25324206
GO:0008610 Lipid biosynthetic process Increases phenotype PMID:28058446
GO:0030262 Apoptotic nuclear changes Increases phenotype PMID:31152826
GO:0044849 Estrous cycle Affects phenotype PMID:37551828
GO:0045795 Positive regulation of cell volume Increases phenotype PMID:29038839
GO:0047823 D-glutamyltransferase activity Increases phenotype PMID:29038839
GO:0050810 Regulation of steroid biosynthetic process Affects phenotype PMID:26781511
GO:0060322 Head development Increases phenotype PMID:31708466
GO:0060765 Regulation of androgen receptor signaling pathway Affects phenotype PMID:25324206
GO:0061370 Testosterone biosynthetic process Affects phenotype PMID:33983380; PMID:37551828
GO:0071578 Zinc ion import across plasma membrane Increases phenotype PMID:31152826
GO:1904077 Negative regulation of estrogen biosynthetic process Increases phenotype PMID:29100959; PMID:29100959; PMID:29501854; PMID:29501854
GO:2000224 Regulation of testosterone biosynthetic process Affects phenotype PMID:22461451
GO:2000225 Negative regulation of testosterone biosynthetic process Increases phenotype PMID:29100959; PMID:29501854; PMID:29501854

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.