Amitraz


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0004197 Cysteine-type endopeptidase activity Increases phenotype PMID:31029719
GO:0004510 Tryptophan 5-monooxygenase activity Increases phenotype PMID:28463726
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:19591892
GO:0008210 Estrogen metabolic process Increases phenotype PMID:28463726
GO:0009449 Gamma-aminobutyric acid biosynthetic process Affects phenotype PMID:26141220
GO:0010942 Positive regulation of cell death Increases phenotype PMID:26141220
GO:0016042 Lipid catabolic process Increases phenotype PMID:31029719
GO:0018158 Protein oxidation Increases phenotype PMID:31029719
GO:0032228 Regulation of synaptic transmission, gabaergic Affects phenotype PMID:26141220
GO:0042420 Dopamine catabolic process Decreases phenotype PMID:28463726
GO:0042422 Norepinephrine catabolic process Decreases phenotype PMID:28463726
GO:0042429 Serotonin catabolic process Decreases phenotype PMID:28463726
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:39489475
GO:0044237 Cellular metabolic process Decreases phenotype PMID:31029719
GO:0050665 Hydrogen peroxide biosynthetic process Increases phenotype PMID:31029719
GO:0051966 Regulation of synaptic transmission, glutamatergic Affects phenotype PMID:26141220
GO:0097054 L-glutamate biosynthetic process Affects phenotype PMID:26141220
GO:2000845 Positive regulation of testosterone secretion Increases phenotype PMID:28463726
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.