Pendimethalin


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000737 Dna catabolic process, endonucleolytic Increases phenotype PMID:34738321
GO:0002693 Positive regulation of cellular extravasation Increases phenotype PMID:30459330
GO:0004035 Alkaline phosphatase activity Increases phenotype PMID:34738321
GO:0004069 L-aspartate:2-oxoglutarate aminotransferase activity Increases phenotype PMID:34738321
GO:0006749 Glutathione metabolic process Increases phenotype PMID:30459330
GO:0010940 Positive regulation of necrotic cell death Increases phenotype PMID:30459330
GO:0010942 Positive regulation of cell death Increases phenotype PMID:40434365
GO:0018158 Protein oxidation Increases phenotype PMID:30459330
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:30459330
GO:0031323 Regulation of cellular metabolic process Affects phenotype PMID:28965233
GO:0034440 Lipid oxidation Increases phenotype PMID:30459330; PMID:34738321
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:30459330
GO:0050867 Positive regulation of cell activation Increases phenotype PMID:30459330
GO:0051091 Positive regulation of dna-binding transcription factor activity Increases phenotype PMID:28973306
GO:1901670 Negative regulation of superoxide dismutase activity Increases phenotype PMID:30459330
GO:1901687 Glutathione derivative biosynthetic process Decreases phenotype PMID:30459330
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.