| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0000212 | Meiotic spindle organization | Affects phenotype | PMID:34676817 |
| GO:0001556 | Oocyte maturation | Affects phenotype | PMID:34676817 |
| GO:0004035 | Alkaline phosphatase activity | Increases phenotype | PMID:28425351 |
| GO:0004069 | L-aspartate:2-oxoglutarate aminotransferase activity | Increases phenotype | PMID:28425351 |
| GO:0005975 | Carbohydrate metabolic process | Affects phenotype | PMID:28425351 |
| GO:0006012 | Galactose metabolic process | Affects phenotype | PMID:28425351 |
| GO:0006014 | D-ribose metabolic process | Affects phenotype | PMID:28425351 |
| GO:0006020 | Inositol metabolic process | Affects phenotype | PMID:28425351 |
| GO:0006560 | Proline metabolic process | Affects phenotype | PMID:28425351 |
| GO:0006633 | Fatty acid biosynthetic process | Affects phenotype | PMID:28425351 |
| GO:0010867 | Positive regulation of triglyceride biosynthetic process | Increases phenotype | PMID:39742927 |
| GO:0010940 | Positive regulation of necrotic cell death | Increases phenotype | PMID:39742927 |
| GO:0010983 | Positive regulation of high-density lipoprotein particle clearance | Increases phenotype | PMID:39742927 |
| GO:0015939 | Pantothenate metabolic process | Affects phenotype | PMID:28425351 |
| GO:0019216 | Regulation of lipid metabolic process | Affects phenotype | PMID:39742927 |
| GO:0019321 | Pentose metabolic process | Affects phenotype | PMID:28425351 |
| GO:0019752 | Carboxylic acid metabolic process | Affects phenotype | PMID:28425351; PMID:38734222 |
| GO:0040038 | Polar body extrusion after meiotic divisions | Affects phenotype | PMID:34676817 |
| GO:0042573 | Retinoic acid metabolic process | Affects phenotype | PMID:28425351 |
| GO:0042732 | D-xylose metabolic process | Affects phenotype | PMID:28425351 |
| GO:0050729 | Positive regulation of inflammatory response | Increases phenotype | PMID:39742927 |
| GO:0050996 | Positive regulation of lipid catabolic process | Increases phenotype | PMID:39742927 |
| GO:0051882 | Mitochondrial depolarization | Increases phenotype | PMID:34676817 |
| GO:0072350 | Tricarboxylic acid metabolic process | Affects phenotype | PMID:28425351 |
| GO:0072593 | Reactive oxygen species metabolic process | Affects phenotype | PMID:34676817 |
| GO:0090306 | Meiotic spindle assembly | Affects phenotype | PMID:34676817 |
| GO:1900533 | Palmitic acid metabolic process | Affects phenotype | PMID:28425351 |
| GO:1901334 | Lactone metabolic process | Affects phenotype | PMID:28425351 |
| GO:1903787 | Negative regulation of glutathione biosynthetic process | Increases phenotype | PMID:39742927 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.