| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0004517 | Nitric-oxide synthase activity | Increases phenotype | PMID:24860958 |
| GO:0004602 | Glutathione peroxidase activity | Increases phenotype | PMID:24860958 |
| GO:0004784 | Superoxide dismutase activity | Increases phenotype | PMID:24860958 |
| GO:0006809 | Nitric oxide biosynthetic process | Increases phenotype | PMID:24860958 |
| GO:0006811 | Monoatomic ion transport | Increases phenotype | PMID:3826359 |
| GO:0008217 | Regulation of blood pressure | Affects phenotype | PMID:20876122 |
| GO:0031443 | Fast-twitch skeletal muscle fiber contraction | Increases phenotype | PMID:22687611 |
| GO:0042593 | Glucose homeostasis | Affects phenotype | PMID:20876122 |
| GO:0043065 | Positive regulation of apoptotic process | Increases phenotype | PMID:10920863; PMID:22484629 |
| GO:0045429 | Positive regulation of nitric oxide biosynthetic process | Increases phenotype | PMID:17275683 |
| GO:0046622 | Positive regulation of organ growth | Increases phenotype | PMID:34409734 |
| GO:0046984 | Regulation of hemoglobin biosynthetic process | Affects phenotype | PMID:24740966 |
| GO:0050665 | Hydrogen peroxide biosynthetic process | Increases phenotype | PMID:19922758; PMID:22484629 |
| GO:0070254 | Mucus secretion | Increases phenotype | PMID:17275683 |
| GO:1901318 | Negative regulation of flagellated sperm motility | Increases phenotype | PMID:19922758 |
| GO:1905221 | Positive regulation of platelet formation | Increases phenotype | PMID:34409734 |
| GO:2000403 | Positive regulation of lymphocyte migration | Increases phenotype | PMID:34409734 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.