| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0002440 | Production of molecular mediator of immune response | Increases phenotype | PMID:18995849 |
| GO:0006915 | Apoptotic process | Increases phenotype | PMID:29704545 |
| GO:0006997 | Nucleus organization | Decreases phenotype | PMID:29704545 |
| GO:0007616 | Long-term memory | Decreases phenotype | PMID:26480858 |
| GO:0008219 | Cell death | Increases phenotype | PMID:30075218 |
| GO:0008361 | Regulation of cell size | Affects phenotype | PMID:29704545 |
| GO:0016049 | Cell growth | Decreases phenotype | PMID:29704545 |
| GO:0030317 | Flagellated sperm motility | Decreases phenotype | PMID:12948891 |
| GO:0032204 | Regulation of telomere maintenance | Affects phenotype | PMID:32300848 |
| GO:0032633 | Interleukin-4 production | Decreases phenotype | PMID:11266329 |
| GO:0042403 | Thyroid hormone metabolic process | Affects phenotype | PMID:19750101 |
| GO:0042445 | Hormone metabolic process | Affects phenotype | PMID:17938741 |
| GO:0044027 | Hypermethylation of cpg island | Affects phenotype | PMID:25585924 |
| GO:0048589 | Developmental growth | Decreases phenotype | PMID:21851983 |
| GO:0050890 | Cognition | Decreases phenotype | PMID:18288320 |
| GO:0070266 | Necroptotic process | Increases phenotype | PMID:29704545 |
| GO:2000612 | Regulation of thyroid-stimulating hormone secretion | Affects phenotype | PMID:19750101 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.