Monosodium glutamate


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001541 Ovarian follicle development Decreases phenotype PMID:30733785
GO:0001552 Ovarian follicle atresia Increases phenotype PMID:30733785
GO:0001556 Oocyte maturation Decreases phenotype PMID:30733785
GO:0001659 Temperature homeostasis Affects phenotype PMID:25211138
GO:0001678 Cellular glucose homeostasis Affects phenotype PMID:21549692
GO:0002024 Diet induced thermogenesis Increases phenotype PMID:2307116
GO:0002064 Epithelial cell development Affects phenotype PMID:22223845
GO:0002523 Leukocyte migration involved in inflammatory response Increases phenotype PMID:21549692; PMID:34560825; PMID:38084638
GO:0004035 Alkaline phosphatase activity Increases phenotype PMID:34560825
GO:0004069 L-aspartate:2-oxoglutarate aminotransferase activity Increases phenotype PMID:18048028; PMID:34560825
GO:0004095 Carnitine o-palmitoyltransferase activity Affects phenotype PMID:15044358
GO:0004111 Creatine kinase activity Increases phenotype PMID:26900785; PMID:26900785; PMID:34560825
GO:0004364 Glutathione transferase activity Affects phenotype PMID:26319613; PMID:29441869
GO:0004457 Lactate dehydrogenase activity Increases phenotype PMID:26900785; PMID:26900785; PMID:34560825
GO:0004602 Glutathione peroxidase activity Affects phenotype PMID:25473020; PMID:26900785; PMID:29679905
GO:0004784 Superoxide dismutase activity Affects phenotype PMID:18048028; PMID:26900785; PMID:29679905; PMID:30733785; PMID:34560825; PMID:38084638
GO:0005391 P-type sodium:potassium-exchanging transporter activity Decreases phenotype PMID:21999347
GO:0006006 Glucose metabolic process Increases phenotype PMID:38084638
GO:0006094 Gluconeogenesis Affects phenotype PMID:11319642; PMID:18048028; PMID:28708105
GO:0006114 Glycerol biosynthetic process Affects phenotype PMID:11319642
GO:0006325 Chromatin organization Increases phenotype PMID:15139026
GO:0006695 Cholesterol biosynthetic process Affects phenotype PMID:18048028; PMID:18048028; PMID:21549692; PMID:19724016; PMID:19724016; PMID:22223845; PMID:22415589; PMID:29679905; PMID:32103741; PMID:19800637; PMID:21549692; PMID:21999347; PMID:22223845; PMID:22415589; PMID:23783067; PMID:25834641; PMID:26092479; PMID:29679905; PMID:32103741
GO:0006749 Glutathione metabolic process Affects phenotype PMID:38084638
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:22447225; PMID:26900785
GO:0006809 Nitric oxide biosynthetic process Increases phenotype PMID:21999347
GO:0006915 Apoptotic process Increases phenotype PMID:22169520; PMID:36732937
GO:0006954 Inflammatory response Increases phenotype PMID:26670228; PMID:32103741
GO:0007033 Vacuole organization Increases phenotype PMID:15139026; PMID:38084638
GO:0007283 Spermatogenesis Increases phenotype PMID:24502529
GO:0007285 Primary spermatocyte growth Affects phenotype PMID:24502529
GO:0007409 Axonogenesis Increases phenotype PMID:30837834
GO:0007631 Feeding behavior Decreases phenotype PMID:12077204; PMID:25473020; PMID:25497710
GO:0008219 Cell death Affects phenotype PMID:12077204; PMID:15044358; PMID:20376352; PMID:29328403; PMID:19093718; PMID:19151514; PMID:19733649; PMID:2004597; PMID:20376352; PMID:29328403; PMID:30837834; PMID:3299706
GO:0008283 Cell population proliferation Increases phenotype PMID:24502529; PMID:25497710
GO:0008361 Regulation of cell size Affects phenotype PMID:15139026
GO:0008610 Lipid biosynthetic process Increases phenotype PMID:11319642; PMID:26670228; PMID:34560825
GO:0009062 Fatty acid catabolic process Affects phenotype PMID:11319642; PMID:18048028
GO:0014009 Glial cell proliferation Increases phenotype PMID:20928830
GO:0015020 Glucuronosyltransferase activity Affects phenotype PMID:25663641
GO:0015734 Taurine transport Decreases phenotype PMID:31569818
GO:0016042 Lipid catabolic process Increases phenotype PMID:21549692; PMID:21999347; PMID:22447225; PMID:26900785; PMID:29679905
GO:0016887 Atp hydrolysis activity Decreases phenotype PMID:21999347
GO:0018158 Protein oxidation Increases phenotype PMID:29679905
GO:0019432 Triglyceride biosynthetic process Increases phenotype PMID:11319642; PMID:18048028; PMID:18048028; PMID:19724016; PMID:21549692; PMID:18048028; PMID:19800637; PMID:21549692; PMID:28708105; PMID:19001666; PMID:19724016; PMID:20111022; PMID:22223845; PMID:22415589; PMID:29679905; PMID:19724016; PMID:19800637; PMID:20111022; PMID:21549692; PMID:21999347; PMID:22223845; PMID:22415589; PMID:23783067; PMID:25211138; PMID:26092479; PMID:26092479; PMID:26670228; PMID:28708105; PMID:29679905
GO:0030263 Apoptotic chromosome condensation Affects phenotype PMID:28954212; PMID:38084638
GO:0030391 Fructosamine biosynthetic process Increases phenotype PMID:28708105
GO:0030431 Sleep Increases phenotype PMID:25697375
GO:0032098 Regulation of appetite Affects phenotype PMID:19093718
GO:0034440 Lipid oxidation Increases phenotype PMID:38084638
GO:0035812 Renal sodium excretion Increases phenotype PMID:31569818
GO:0035930 Corticosteroid hormone secretion Increases phenotype PMID:31077719
GO:0035934 Corticosterone secretion Increases phenotype PMID:8071649
GO:0035936 Testosterone secretion Decreases phenotype PMID:14657608; PMID:24502529
GO:0035937 Estrogen secretion Decreases phenotype PMID:30733785
GO:0035941 Androstenedione secretion Decreases phenotype PMID:14657608
GO:0036359 Renal potassium excretion Increases phenotype PMID:31569818
GO:0042632 Cholesterol homeostasis Decreases phenotype PMID:31981724
GO:0042701 Progesterone secretion Decreases phenotype PMID:14657608
GO:0043005 Neuron projection Decreases phenotype PMID:39581299
GO:0044237 Cellular metabolic process Decreases phenotype PMID:28954212; PMID:34560825; PMID:36732937; PMID:39581299
GO:0045852 Ph elevation Increases phenotype PMID:31569818
GO:0048143 Astrocyte activation Increases phenotype PMID:20928830
GO:0048874 Host-mediated regulation of intestinal microbiota composition Affects phenotype PMID:32103741
GO:0051092 Positive regulation of nf-kappab transcription factor activity Increases phenotype PMID:22169520
GO:0051790 Short-chain fatty acid biosynthetic process Affects phenotype PMID:32103741
GO:0051939 Gamma-aminobutyric acid import Decreases phenotype PMID:19733649
GO:0060612 Adipose tissue development Increases phenotype PMID:15809509; PMID:18048028; PMID:19093718; PMID:19093718; PMID:19724016; PMID:20111022; PMID:20111022; PMID:21549692; PMID:21549692; PMID:25497710; PMID:3299706
GO:0060613 Fat pad development Increases phenotype PMID:22415589; PMID:26092479
GO:0061900 Glial cell activation Increases phenotype PMID:30837834
GO:0070265 Necrotic cell death Increases phenotype PMID:34560825; PMID:38084638
GO:0070342 Brown fat cell proliferation Decreases phenotype PMID:25211138
GO:0070343 White fat cell proliferation Increases phenotype PMID:25211138; PMID:29328403
GO:0070994 Detection of oxidative stress Affects phenotype PMID:34560825
GO:0070997 Neuron death Increases phenotype PMID:12193538; PMID:15139026; PMID:9053782
GO:0072520 Seminiferous tubule development Affects phenotype PMID:24502529
GO:0140042 Lipid droplet formation Increases phenotype PMID:19001666; PMID:19724016; PMID:25211138
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:29679905
GO:1905517 Macrophage migration Increases phenotype PMID:19800637; PMID:32103741
GO:2000253 Positive regulation of feeding behavior Increases phenotype PMID:12193538

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.