| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0001892 | Embryonic placenta development | Decreases phenotype | PMID:33603097 |
| GO:0006954 | Inflammatory response | Increases phenotype | PMID:23717615 |
| GO:0007204 | Positive regulation of cytosolic calcium ion concentration | Increases phenotype | PMID:27939335 |
| GO:0007613 | Memory | Decreases phenotype | PMID:27128166 |
| GO:0008016 | Regulation of heart contraction | Affects phenotype | PMID:26276052 |
| GO:0008217 | Regulation of blood pressure | Affects phenotype | PMID:26276052 |
| GO:0010467 | Gene expression | Affects phenotype | PMID:25564368 |
| GO:0010468 | Regulation of gene expression | Affects phenotype | PMID:27809917 |
| GO:0010508 | Positive regulation of autophagy | Increases phenotype | PMID:25979628 |
| GO:0018159 | Peptidyl-methionine oxidation | Increases phenotype | PMID:25979628 |
| GO:0030216 | Keratinocyte differentiation | Affects phenotype | PMID:31791492 |
| GO:0032731 | Positive regulation of interleukin-1 beta production | Increases phenotype | PMID:26276052 |
| GO:0044319 | Wound healing, spreading of cells | Increases phenotype | PMID:26880698 |
| GO:0046466 | Membrane lipid catabolic process | Increases phenotype | PMID:34417545 |
| GO:0060047 | Heart contraction | Affects phenotype | PMID:20595912 |
| GO:0060326 | Cell chemotaxis | Increases phenotype | PMID:26880698 |
| GO:0060964 | Regulation of mirna-mediated gene silencing | Affects phenotype | PMID:26068961 |
| GO:0070374 | Positive regulation of erk1 and erk2 cascade | Increases phenotype | PMID:25979628 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.