Abamectin


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000045 Autophagosome assembly Increases phenotype PMID:30958899
GO:0004021 L-alanine:2-oxoglutarate aminotransferase activity Increases phenotype PMID:31739238
GO:0004035 Alkaline phosphatase activity Increases phenotype PMID:31739238
GO:0004069 L-aspartate:2-oxoglutarate aminotransferase activity Increases phenotype PMID:31739238
GO:0004364 Glutathione transferase activity Decreases phenotype PMID:30091233
GO:0006749 Glutathione metabolic process Increases phenotype PMID:31739238; PMID:34748272
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:19591892
GO:0007009 Plasma membrane organization Affects phenotype PMID:34417632
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:29555536; PMID:32201246
GO:0010942 Positive regulation of cell death Increases phenotype PMID:25257666; PMID:34417632
GO:0016042 Lipid catabolic process Affects phenotype PMID:31739238
GO:0030336 Negative regulation of cell migration Increases phenotype PMID:32201246
GO:0032781 Positive regulation of atp-dependent activity Increases phenotype PMID:34417632
GO:0034440 Lipid oxidation Increases phenotype PMID:34748272
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:32201246
GO:0044237 Cellular metabolic process Decreases phenotype PMID:30958899
GO:0045023 G0 to g1 transition Increases phenotype PMID:30958899
GO:0045930 Negative regulation of mitotic cell cycle Increases phenotype PMID:30958899
GO:0051881 Regulation of mitochondrial membrane potential Affects phenotype PMID:30958899
GO:0070633 Transepithelial transport Increases phenotype PMID:34417632
GO:1901670 Negative regulation of superoxide dismutase activity Increases phenotype PMID:34748272
GO:1902476 Chloride transmembrane transport Increases phenotype PMID:32201246
GO:1903283 Negative regulation of glutathione peroxidase activity Increases phenotype PMID:34748272
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:30958899
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.