Benzophenone-3


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001787 Natural killer cell proliferation Increases phenotype PMID:38431166
GO:0001892 Embryonic placenta development Affects phenotype PMID:38431166
GO:0001944 Vasculature development Decreases phenotype PMID:39343157
GO:0002043 Blood vessel endothelial cell proliferation involved in sprouting angiogenesis Decreases phenotype PMID:39343157
GO:0003014 Renal system process Decreases phenotype PMID:30660848
GO:0006309 Apoptotic dna fragmentation Increases phenotype PMID:30099065
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:31939680
GO:0006979 Response to oxidative stress Affects phenotype PMID:31368502
GO:0007566 Embryo implantation Affects phenotype PMID:38431166
GO:0007613 Memory Decreases phenotype PMID:31368502
GO:0008219 Cell death Increases phenotype PMID:30391638
GO:0008283 Cell population proliferation Affects phenotype PMID:28855101; PMID:31408669; PMID:35669359; PMID:39343157
GO:0008361 Regulation of cell size Affects phenotype PMID:35669359
GO:0010424 Dna methylation on cytosine within a cg sequence Affects phenotype PMID:32755991; PMID:34523531
GO:0010719 Negative regulation of epithelial to mesenchymal transition Increases phenotype PMID:38387520
GO:0030336 Negative regulation of cell migration Increases phenotype PMID:38387520
GO:0030879 Mammary gland development Decreases phenotype PMID:31408669
GO:0033023 Mast cell homeostasis Affects phenotype PMID:35669359
GO:0033148 Positive regulation of intracellular estrogen receptor signaling pathway Increases phenotype PMID:25449125; PMID:31939680
GO:0036099 Female germ-line stem cell population maintenance Decreases phenotype PMID:30599193
GO:0042695 Thelarche Decreases phenotype PMID:26335517
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:31368502
GO:0044319 Wound healing, spreading of cells Decreases phenotype PMID:39343157
GO:0046884 Follicle-stimulating hormone secretion Decreases phenotype PMID:30245359
GO:0048160 Primary follicle stage Decreases phenotype PMID:30599193
GO:0060138 Fetal process involved in parturition Affects phenotype PMID:38431166
GO:0060322 Head development Increases phenotype PMID:21900077
GO:0060612 Adipose tissue development Decreases phenotype PMID:27037776
GO:0061370 Testosterone biosynthetic process Decreases phenotype PMID:27383665; PMID:30099065; PMID:30316929
GO:0071578 Zinc ion import across plasma membrane Increases phenotype PMID:30391638
GO:0140042 Lipid droplet formation Increases phenotype PMID:32407875; PMID:34864131
GO:1903294 Regulation of glutamate secretion, neurotransmission Affects phenotype PMID:31368502
GO:1905938 Positive regulation of germ cell proliferation Increases phenotype PMID:30599193
GO:2000845 Positive regulation of testosterone secretion Increases phenotype PMID:30245359
GO:2000866 Positive regulation of estradiol secretion Increases phenotype PMID:30245359
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.