| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0002523 | Leukocyte migration involved in inflammatory response | Increases phenotype | PMID:37156404 |
| GO:0006006 | Glucose metabolic process | Affects phenotype | PMID:37156404 |
| GO:0006641 | Triglyceride metabolic process | Affects phenotype | PMID:37156404 |
| GO:0008203 | Cholesterol metabolic process | Affects phenotype | PMID:37156404 |
| GO:0008283 | Cell population proliferation | Decreases phenotype | PMID:28709983 |
| GO:0016505 | Peptidase activator activity involved in apoptotic process | Increases phenotype | PMID:28709983 |
| GO:0019627 | Urea metabolic process | Affects phenotype | PMID:37156404 |
| GO:0044255 | Cellular lipid metabolic process | Affects phenotype | PMID:34323617 |
| GO:0045444 | Fat cell differentiation | Increases phenotype | PMID:37142754 |
| GO:0140042 | Lipid droplet formation | Increases phenotype | PMID:37142754; PMID:37156404 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.