| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0006306 | Dna methylation | Affects phenotype | PMID:26339649 |
| GO:0006641 | Triglyceride metabolic process | Affects phenotype | PMID:31835022 |
| GO:0008203 | Cholesterol metabolic process | Affects phenotype | PMID:31835022 |
| GO:0010868 | Negative regulation of triglyceride biosynthetic process | Increases phenotype | PMID:34038317 |
| GO:0031507 | Heterochromatin formation | Decreases phenotype | PMID:26339649 |
| GO:0032502 | Developmental process | Decreases phenotype | PMID:27153333 |
| GO:0042593 | Glucose homeostasis | Decreases phenotype | PMID:35777568 |
| GO:0044030 | Regulation of dna methylation | Increases phenotype | PMID:37914285 |
| GO:0045666 | Positive regulation of neuron differentiation | Increases phenotype | PMID:37914285 |
| GO:0045792 | Negative regulation of cell size | Increases phenotype | PMID:37914285 |
| GO:0048589 | Developmental growth | Decreases phenotype | PMID:31708466 |
| GO:0050890 | Cognition | Decreases phenotype | PMID:27153333 |
| GO:0051460 | Negative regulation of corticotropin secretion | Increases phenotype | PMID:35777568 |
| GO:0051463 | Negative regulation of cortisol secretion | Increases phenotype | PMID:35777568 |
| GO:0060322 | Head development | Decreases phenotype | PMID:27153333; PMID:31708466 |
| GO:1901142 | Insulin metabolic process | Affects phenotype | PMID:35777568 |
| GO:2000844 | Negative regulation of testosterone secretion | Increases phenotype | PMID:34038317 |
| GO:2000853 | Negative regulation of corticosterone secretion | Increases phenotype | PMID:35777568 |
| GO:2000866 | Positive regulation of estradiol secretion | Increases phenotype | PMID:34038317 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.