Vinyl chloride


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001510 Rna methylation Affects phenotype PMID:37061008
GO:0006695 Cholesterol biosynthetic process Increases phenotype PMID:31445928
GO:0006749 Glutathione metabolic process Increases phenotype PMID:34717031
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:31445928
GO:0006954 Inflammatory response Increases phenotype PMID:26962056
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:37061008
GO:0010468 Regulation of gene expression Affects phenotype PMID:36892438
GO:0010942 Positive regulation of cell death Increases phenotype PMID:26962056
GO:0016042 Lipid catabolic process Increases phenotype PMID:31445928
GO:0019432 Triglyceride biosynthetic process Increases phenotype PMID:31445928
GO:0043029 T cell homeostasis Decreases phenotype PMID:34717031
GO:0051881 Regulation of mitochondrial membrane potential Affects phenotype PMID:26962056
GO:0061484 Hematopoietic stem cell homeostasis Decreases phenotype PMID:34717031
GO:0061519 Macrophage homeostasis Decreases phenotype PMID:34717031
GO:0140042 Lipid droplet formation Increases phenotype PMID:31445928
GO:1900017 Positive regulation of cytokine production involved in inflammatory response Increases phenotype PMID:31774537
GO:1904598 Positive regulation of connective tissue replacement involved in inflammatory response wound healing Increases phenotype PMID:37061008
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.