| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0001516 | Prostaglandin biosynthetic process | Increases phenotype | PMID:22155309 |
| GO:0003094 | Glomerular filtration | Decreases phenotype | PMID:2203262 |
| GO:0005980 | Glycogen catabolic process | Increases phenotype | PMID:7713362 |
| GO:0006693 | Prostaglandin metabolic process | Affects phenotype | PMID:25034942 |
| GO:0006749 | Glutathione metabolic process | Affects phenotype | PMID:25034942 |
| GO:0006915 | Apoptotic process | Increases phenotype | PMID:12357301 |
| GO:0006979 | Response to oxidative stress | Increases phenotype | PMID:16472794; PMID:9583089 |
| GO:0007049 | Cell cycle | Affects phenotype | PMID:12357301 |
| GO:0007283 | Spermatogenesis | Decreases phenotype | PMID:25034942 |
| GO:0008217 | Regulation of blood pressure | Affects phenotype | PMID:12152004; PMID:8227469; PMID:9566780 |
| GO:0009636 | Response to toxic substance | Decreases phenotype | PMID:21319177 |
| GO:0010942 | Positive regulation of cell death | Increases phenotype | PMID:28754375 |
| GO:0035812 | Renal sodium excretion | Decreases phenotype | PMID:12152004 |
| GO:0042178 | Xenobiotic catabolic process | Affects phenotype | PMID:16472794 |
| GO:0046466 | Membrane lipid catabolic process | Increases phenotype | PMID:7718302; PMID:9583089 |
| GO:0060326 | Cell chemotaxis | Decreases phenotype | PMID:314231; PMID:6853143 |
| GO:0097722 | Sperm motility | Decreases phenotype | PMID:25034942 |
| GO:1904996 | Positive regulation of leukocyte adhesion to vascular endothelial cell | Increases phenotype | PMID:14762100 |
| GO:2001171 | Positive regulation of atp biosynthetic process | Decreases phenotype | PMID:28754375 |
| GO:2001303 | Lipoxin a4 biosynthetic process | Increases phenotype | PMID:14762100 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.