| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0001556 | Oocyte maturation | Decreases phenotype | PMID:26545148 |
| GO:0003016 | Respiratory system process | Decreases phenotype | PMID:27852937; PMID:30737192 |
| GO:0003093 | Regulation of glomerular filtration | Affects phenotype | PMID:32645486 |
| GO:0006590 | Thyroid hormone generation | Increases phenotype | PMID:31093596 |
| GO:0006936 | Muscle contraction | Decreases phenotype | PMID:27581612 |
| GO:0007283 | Spermatogenesis | Decreases phenotype | PMID:18570050 |
| GO:0007628 | Adult walking behavior | Affects phenotype | PMID:37018009 |
| GO:0010424 | Dna methylation on cytosine within a cg sequence | Affects phenotype | PMID:37120575 |
| GO:0018963 | Phthalate metabolic process | Increases phenotype | PMID:35354077 |
| GO:0034599 | Cellular response to oxidative stress | Increases phenotype | PMID:38503103 |
| GO:0043396 | Corticotropin-releasing hormone secretion | Decreases phenotype | PMID:31093596 |
| GO:0046339 | Diacylglycerol metabolic process | Affects phenotype | PMID:35075242 |
| GO:0046544 | Development of secondary male sexual characteristics | Decreases phenotype | PMID:24945889 |
| GO:0060322 | Head development | Affects phenotype | PMID:25014249 |
| GO:0090181 | Regulation of cholesterol metabolic process | Affects phenotype | PMID:37093692 |
| GO:0140747 | Regulation of ncrna transcription | Affects phenotype | PMID:37003585 |
| GO:1901318 | Negative regulation of flagellated sperm motility | Increases phenotype | PMID:24140385 |
| GO:1990428 | Mirna transport | Affects phenotype | PMID:30481674 |
| GO:2000609 | Regulation of thyroid hormone generation | Affects phenotype | PMID:32464474 |
| GO:2000610 | Negative regulation of thyroid hormone generation | Affects phenotype | PMID:34708313; PMID:38042133 |
| GO:2000611 | Positive regulation of thyroid hormone generation | Affects phenotype | PMID:34708313 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.