Captan


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000104 Succinate dehydrogenase activity Decreases phenotype PMID:10561079
GO:0000737 Dna catabolic process, endonucleolytic Increases phenotype PMID:34303901
GO:0001541 Ovarian follicle development Affects phenotype PMID:34303901
GO:0001552 Ovarian follicle atresia Increases phenotype PMID:34303901
GO:0004030 Aldehyde dehydrogenase [nad(p)+] activity Decreases phenotype PMID:24491970
GO:0006006 Glucose metabolic process Affects phenotype PMID:32745781
GO:0006083 Acetate metabolic process Affects phenotype PMID:32745781
GO:0006105 Succinate metabolic process Affects phenotype PMID:32745781
GO:0006631 Fatty acid metabolic process Affects phenotype PMID:32745781
GO:0006915 Apoptotic process Increases phenotype PMID:34303901
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:19591892
GO:0007005 Mitochondrion organization Decreases phenotype PMID:34303901
GO:0008203 Cholesterol metabolic process Affects phenotype PMID:32745781
GO:0008283 Cell population proliferation Increases phenotype PMID:32736067
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:32949729
GO:0019627 Urea metabolic process Affects phenotype PMID:32745781
GO:0031987 Locomotion involved in locomotory behavior Decreases phenotype PMID:32736067
GO:0035176 Social behavior Affects phenotype PMID:32736067
GO:0044237 Cellular metabolic process Decreases phenotype PMID:32870474
GO:0046034 Atp metabolic process Affects phenotype PMID:32949729
GO:0050872 White fat cell differentiation Increases phenotype PMID:32745781
GO:0051646 Mitochondrion localization Affects phenotype PMID:34303901
GO:0051882 Mitochondrial depolarization Increases phenotype PMID:34303901
GO:0070994 Detection of oxidative stress Increases phenotype PMID:34303901
GO:1903427 Negative regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:35395341
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.