| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0001569 | Branching involved in blood vessel morphogenesis | Decreases phenotype | PMID:31704167 |
| GO:0007283 | Spermatogenesis | Decreases phenotype | PMID:29447956 |
| GO:0008283 | Cell population proliferation | Decreases phenotype | PMID:28214531 |
| GO:0009790 | Embryo development | Affects phenotype | PMID:31704167; PMID:32622971 |
| GO:0030282 | Bone mineralization | Decreases phenotype | PMID:28214531 |
| GO:0032964 | Collagen biosynthetic process | Decreases phenotype | PMID:28214531 |
| GO:0035936 | Testosterone secretion | Affects phenotype | PMID:34958886 |
| GO:0040015 | Negative regulation of multicellular organism growth | Increases phenotype | PMID:29447956 |
| GO:0042632 | Cholesterol homeostasis | Decreases phenotype | PMID:29447956 |
| GO:0046621 | Negative regulation of organ growth | Increases phenotype | PMID:29447956 |
| GO:0098868 | Bone growth | Decreases phenotype | PMID:28214531 |
| GO:1901318 | Negative regulation of flagellated sperm motility | Increases phenotype | PMID:29447956 |
| GO:2000225 | Negative regulation of testosterone biosynthetic process | Increases phenotype | PMID:29447956 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.