Boscalid


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0006006 Glucose metabolic process Affects phenotype PMID:32745781
GO:0006083 Acetate metabolic process Affects phenotype PMID:32745781
GO:0006105 Succinate metabolic process Affects phenotype PMID:32745781
GO:0006631 Fatty acid metabolic process Affects phenotype PMID:32745781
GO:0006641 Triglyceride metabolic process Affects phenotype PMID:32810590
GO:0008203 Cholesterol metabolic process Affects phenotype PMID:32745781
GO:0008283 Cell population proliferation Affects phenotype PMID:28855101; PMID:32736067
GO:0018411 Protein glucuronidation Increases phenotype PMID:36549901
GO:0019627 Urea metabolic process Affects phenotype PMID:32745781
GO:0031393 Negative regulation of prostaglandin biosynthetic process Increases phenotype PMID:26359731
GO:0031987 Locomotion involved in locomotory behavior Decreases phenotype PMID:32736067
GO:0035176 Social behavior Affects phenotype PMID:32736067
GO:0050872 White fat cell differentiation Increases phenotype PMID:32745781

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.