alpha-Naphthoflavone


Curated chemical-gene interactions from CTD
Entrez gene IDGene symbolInteraction typeReference
140634 CYP1A Decreases activity PMID:15916741; PMID:8819299
1543 CYP1A1 Affects activity PMID:11516172; PMID:12859983; PMID:1329656; PMID:15276085; PMID:15306208; PMID:15456928; PMID:15766918; PMID:15893423; PMID:16426572; PMID:21053930; PMID:6477826; PMID:16906435
1543 CYP1A1 Affects expression PMID:11516172; PMID:12859983; PMID:30508555; PMID:1329656; PMID:17069994; PMID:27163632; PMID:15698582; PMID:19828881; PMID:21127131; PMID:21162728; PMID:21127131; PMID:30508555
1544 CYP1A2 Decreases activity PMID:15306208; PMID:15766918; PMID:15860655; PMID:21053930; PMID:26599973; PMID:29074051; PMID:33253783; PMID:33438235; PMID:21766881; PMID:28887089
1544 CYP1A2 Increases expression PMID:19828881; PMID:20448087; PMID:28887089
1545 CYP1B1 Affects expression PMID:22262711; PMID:39653182
1545 CYP1B1 Decreases activity PMID:15280921; PMID:19563207; PMID:21053930
1573 CYP2J2 Decreases activity PMID:12851038
1576 CYP3A4 Affects activity PMID:15005856; PMID:21053930; PMID:2271712; PMID:31756459
1584 CYP11B1 Increases expression PMID:22172629
1586 CYP17A1 Decreases expression PMID:24875446
1588 CYP19A1 Decreases activity PMID:9435150
1588 CYP19A1 Increases expression PMID:20823264
1649 DDIT3 Increases expression PMID:30508555
1728 NQO1 Increases activity PMID:16906435
1728 NQO1 Increases expression PMID:17069994
17748 MT1 Increases expression PMID:14998373
196 AHR Affects activity PMID:11165043; PMID:11768231; PMID:14993811; PMID:15019843; PMID:16054781; PMID:8390116; PMID:11516172; PMID:11516172; PMID:26519956; PMID:12051995; PMID:12093619; PMID:12111005; PMID:15364539; PMID:17681673; PMID:12559965; PMID:15459018; PMID:15041469; PMID:15797623; PMID:17669606; PMID:8738555; PMID:15201442; PMID:19828881; PMID:20634293; PMID:21127131; PMID:8937476; PMID:9176739
196 AHR Decreases expression PMID:11516172
25976 TIPARP Decreases expression PMID:18762178
2661 GDF9 Increases expression PMID:29094188
2821 GPI Increases expression PMID:23152189
2950 GSTP1 Decreases expression PMID:18762178
3098 HK1 Increases expression PMID:23152189
3487 IGFBP4 Increases expression PMID:23639626
3553 IL1B Affects expression PMID:22262711
3557 IL1RN Increases expression PMID:22262711
3569 IL6 Increases expression PMID:21162728
3576 CXCL8 Affects expression PMID:22262711
367 AR Increases expression PMID:15041469
3725 JUN Increases expression PMID:15897893
3939 LDHA Increases expression PMID:23152189
405 ARNT Increases activity PMID:21127131
4288 MKI67 Increases expression PMID:39653182
4843 NOS2 Increases expression PMID:33722737
5055 SERPINB2 Affects expression PMID:22262711
5213 PFKM Increases expression PMID:23152189
5230 PGK1 Increases expression PMID:23152189
5236 PGM1 Increases expression PMID:23152189
5241 PGR Increases expression PMID:22266097
5743 PTGS2 Affects expression PMID:21162728; PMID:22262711; PMID:22262711
5970 RELA Decreases expression PMID:33722737
6347 CCL2 Affects expression PMID:22262711
6513 SLC2A1 Increases expression PMID:23152189
669 BPGM Increases expression PMID:23152189
6932 TCF7 Increases expression PMID:18762178
7157 TP53 Increases expression PMID:29274324
7167 TPI1 Increases expression PMID:23152189
836 CASP3 Affects activity PMID:15710172; PMID:18334599
841 CASP8 Increases activity PMID:18334599
842 CASP9 Increases activity PMID:18334599
9970 NR1I3 Increases activity PMID:27732639

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.