Lead chloride


Curated chemical-gene interactions from CTD
Entrez gene IDGene symbolInteraction typeReference
10226 PLIN3 Decreases expression PMID:24419708
10383 TUBB4B Increases expression PMID:24419708
10576 CCT2 Decreases expression PMID:24419708
13087 CYP2A5 Increases expression PMID:20402460
1543 CYP1A1 Increases expression PMID:23503628
1645 AKR1C1 Increases expression PMID:18654764
1646 AKR1C2 Increases expression PMID:18654764
1728 NQO1 Increases expression PMID:38447685
17748 MT1 Increases expression PMID:23503628
17750 MT2 Increases expression PMID:23503628
1933 EEF1B2 Increases expression PMID:24419708
1936 EEF1D Decreases expression PMID:24419708
2023 ENO1 Increases expression PMID:24419708
203068 TUBB Increases expression PMID:24419708
2099 ESR1 Decreases expression PMID:12746304
2099 ESR1 Increases activity PMID:12746304
22948 CCT5 Decreases expression PMID:24419708
23564 DDAH2 Increases expression PMID:24419708
2563 GABRD Increases expression PMID:19651682
2670 GFAP Decreases expression PMID:19651682
2729 GCLC Affects expression PMID:18654764; PMID:30359721
2730 GCLM Increases expression PMID:38447685
2877 GPX2 Increases expression PMID:23503628
28984 RGCC Increases expression PMID:18654764
2902 GRIN1 Increases expression PMID:19651682
2923 PDIA3 Decreases expression PMID:24419708
2936 GSR Increases expression PMID:38447685
2944 GSTM1 Decreases expression PMID:30359721
301 ANXA1 Increases expression PMID:24419708
3145 HMBS Decreases activity PMID:10416826
3162 HMOX1 Affects expression PMID:20402460; PMID:23916686; PMID:23503628; PMID:38447685
3309 HSPA5 Affects expression PMID:19720107; PMID:21073006; PMID:24419708
3313 HSPA9 Decreases expression PMID:24419708
3315 HSPB1 Increases expression PMID:24419708
3329 HSPD1 Decreases expression PMID:24419708
3458 IFNG Decreases expression PMID:15843504; PMID:23503628
3553 IL1B Decreases expression PMID:15843504; PMID:23503628
3558 IL2 Decreases expression PMID:15183152
4069 LYZ Decreases expression PMID:24419708
4489 MT1A Increases expression PMID:19720107
4637 MYL6 Increases expression PMID:24419708
4684 NCAM1 Decreases expression PMID:18499259
4688 NCF2 Increases expression PMID:38447685
4744 NEFH Increases expression PMID:19651682
4780 NFE2L2 Decreases expression PMID:30359721
4842 NOS1 Affects expression PMID:24881885; PMID:25410796
4843 NOS2 Affects expression PMID:23503628; PMID:23916686; PMID:25410796
4846 NOS3 Decreases expression PMID:25410796
4891 SLC11A2 Increases expression PMID:23503628
50507 NOX4 Increases expression PMID:30359721
5241 PGR Increases expression PMID:12746304
5599 MAPK8 Increases expression PMID:24419708
5601 MAPK9 Increases expression PMID:24419708
57016 AKR1B10 Increases expression PMID:18654764
596 BCL2 Decreases expression PMID:24419708
60 ACTB Decreases expression PMID:24419708
6038 RNASE4 Increases expression PMID:18654764
6282 S100A11 Increases expression PMID:24419708
6647 SOD1 Affects expression PMID:23503628
6696 SPP1 Increases expression PMID:38447685
7040 TGFB1 Decreases expression PMID:23503628
7086 TKT Increases expression PMID:18654764
7102 TSPAN7 Increases expression PMID:18654764
7124 TNF Decreases expression PMID:15843504; PMID:23503628
7415 VCP Decreases expression PMID:24419708
7431 VIM Decreases expression PMID:24419708
7903 ST8SIA4 Decreases expression PMID:18499259
800 CALD1 Decreases expression PMID:24419708
8128 ST8SIA2 Decreases expression PMID:18499259
90550 MCU Decreases expression PMID:24881885
9376 SLC22A8 Increases activity PMID:28629854

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.