Perfluorooctanesulfonamide


Curated chemical-gene interactions from CTD
Entrez gene IDGene symbolInteraction typeReference
1003 CDH5 Affects expression PMID:33677611
10599 SLCO1B1 Increases expression PMID:35337807; PMID:38953992
10965 ACOT2 Increases expression PMID:35337807
113868 ACAA1A Increases activity PMID:15071170
113868 ACAA1A Increases expression PMID:15071170
123264 SLC51B Decreases expression PMID:38953992
1581 CYP7A1 Increases expression PMID:35337807
1593 CYP27A1 Decreases expression PMID:38953992
1728 NQO1 Increases expression PMID:35337807
19 ABCA1 Increases expression PMID:35337807
1962 EHHADH Increases activity PMID:15071170
1962 EHHADH Increases expression PMID:15071170; PMID:35337807
2194 FASN Increases expression PMID:35337807
2938 GSTA1 Increases expression PMID:35337807
2947 GSTM3 Increases expression PMID:35337807
3157 HMGCS1 Increases expression PMID:35337807
3172 HNF4A Decreases expression PMID:38953992
3672 ITGA1 Affects expression PMID:33677611
39 ACAT2 Decreases expression PMID:38953992
4312 MMP1 Affects expression PMID:33677611
4547 MTTP Increases expression PMID:35337807
4780 NFE2L2 Increases expression PMID:35337807
5106 PCK2 Increases expression PMID:35337807
5465 PPARA Affects activity PMID:15071170; PMID:28857218
5465 PPARA Affects expression PMID:15071170; PMID:35337807; PMID:38953992
54658 UGT1A1 Increases expression PMID:38953992
5467 PPARD Increases expression PMID:38953992
56898 BDH2 Increases expression PMID:35337807
57678 GPAM Increases expression PMID:35337807
6319 SCD Increases expression PMID:35337807
6364 CCL20 Increases expression PMID:38953992
6647 SOD1 Increases expression PMID:35337807
6720 SREBF1 Increases expression PMID:35337807
6822 SULT2A1 Increases expression PMID:35337807
8647 ABCB11 Decreases expression PMID:38953992
8856 NR1I2 Affects expression PMID:35337807; PMID:38953992
9970 NR1I3 Increases expression PMID:35337807
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.