Isoflurane


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000165 Mapk cascade Increases phenotype PMID:18349187
GO:0002027 Regulation of heart rate Increases phenotype PMID:10051936; PMID:10051936; PMID:11125686; PMID:3605747; PMID:3661938; PMID:3994880; PMID:6391530; PMID:7743570; PMID:1308378; PMID:3284271; PMID:7511357; PMID:7717564; PMID:7743570; PMID:8614295
GO:0002420 Natural killer cell mediated cytotoxicity directed against tumor cell target Decreases phenotype PMID:27940100
GO:0002433 Immune response-regulating cell surface receptor signaling pathway involved in phagocytosis Decreases phenotype PMID:28003439
GO:0004197 Cysteine-type endopeptidase activity Increases phenotype PMID:32663520
GO:0004457 Lactate dehydrogenase activity Increases phenotype PMID:33508982; PMID:35164634
GO:0004602 Glutathione peroxidase activity Decreases phenotype PMID:28084090
GO:0004784 Superoxide dismutase activity Decreases phenotype PMID:28084090; PMID:28084090; PMID:35164634; PMID:32663520; PMID:32663520; PMID:35164634; PMID:33508982; PMID:35164634
GO:0006915 Apoptotic process Affects phenotype PMID:11748407; PMID:14980940; PMID:15915027; PMID:16249675; PMID:16827187; PMID:17234824; PMID:17287498; PMID:18349187; PMID:20003127; PMID:25597859; PMID:32663520; PMID:32663520; PMID:35164634; PMID:33508982; PMID:33586613; PMID:35164634; PMID:35187972
GO:0006919 Activation of cysteine-type endopeptidase activity involved in apoptotic process Affects phenotype PMID:23922164
GO:0007009 Plasma membrane organization Decreases phenotype PMID:29289695
GO:0007268 Chemical synaptic transmission Affects phenotype PMID:1317634
GO:0007613 Memory Affects phenotype PMID:33878909
GO:0007614 Short-term memory Decreases phenotype PMID:20966663; PMID:30762015
GO:0007615 Anesthesia-resistant memory Decreases phenotype PMID:20966663
GO:0008217 Regulation of blood pressure Increases phenotype PMID:11230718; PMID:12651645; PMID:1308378; PMID:15297469; PMID:18339214; PMID:1974748; PMID:3284271; PMID:3605747; PMID:6391530; PMID:7486177; PMID:7717564; PMID:8363068; PMID:3284271; PMID:7511357; PMID:7743570; PMID:8614295
GO:0008219 Cell death Increases phenotype PMID:11753020
GO:0008283 Cell population proliferation Affects phenotype PMID:27562558; PMID:27940100
GO:0008284 Positive regulation of cell population proliferation Decreases phenotype PMID:34553399
GO:0008285 Negative regulation of cell population proliferation Affects phenotype PMID:23922164
GO:0008291 Acetylcholine metabolic process Affects phenotype PMID:28084090
GO:0008542 Visual learning Decreases phenotype PMID:33878909
GO:0010508 Positive regulation of autophagy Increases phenotype PMID:34553399
GO:0016042 Lipid catabolic process Affects phenotype PMID:28084090; PMID:32663520
GO:0030593 Neutrophil chemotaxis Decreases phenotype PMID:28003439
GO:0036343 Psychomotor behavior Decreases phenotype PMID:32062618
GO:0042307 Positive regulation of protein import into nucleus Increases phenotype PMID:29289695
GO:0042310 Vasoconstriction Increases phenotype PMID:1319121; PMID:2751124; PMID:7762854; PMID:8001219; PMID:8694307; PMID:8712443; PMID:8712451; PMID:8741470
GO:0042311 Vasodilation Increases phenotype PMID:11230718; PMID:1319121; PMID:2751124; PMID:15297469; PMID:15675976; PMID:1599087; PMID:18339214; PMID:7586449; PMID:7880215; PMID:8624010; PMID:9024015; PMID:9366472
GO:0042391 Regulation of membrane potential Affects phenotype PMID:8614295
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:34553399; PMID:36027947
GO:0043525 Positive regulation of neuron apoptotic process Increases phenotype PMID:23922164; PMID:29289695
GO:0044237 Cellular metabolic process Decreases phenotype PMID:32663520; PMID:32663520; PMID:35164634; PMID:33508982; PMID:33586613; PMID:35164634
GO:0046467 Membrane lipid biosynthetic process Increases phenotype PMID:36027947
GO:0048666 Neuron development Decreases phenotype PMID:27562558
GO:0050890 Cognition Decreases phenotype PMID:28084090; PMID:28084090; PMID:31672664; PMID:31672664; PMID:33586613; PMID:33586613; PMID:35164634; PMID:36000339; PMID:36263453; PMID:35164634; PMID:35187972; PMID:36000339; PMID:36027947; PMID:36263453
GO:0051881 Regulation of mitochondrial membrane potential Decreases phenotype PMID:15915027
GO:0051898 Negative regulation of protein kinase b signaling Affects phenotype PMID:23922164
GO:0051964 Negative regulation of synapse assembly Increases phenotype PMID:35249202
GO:0060079 Excitatory postsynaptic potential Affects phenotype PMID:1317634; PMID:17898380; PMID:15108983; PMID:18362600
GO:0060080 Inhibitory postsynaptic potential Affects phenotype PMID:18362600
GO:0070050 Neuron cellular homeostasis Affects phenotype PMID:35187972
GO:0070269 Pyroptosis Increases phenotype PMID:35187972
GO:0070585 Protein localization to mitochondrion Affects phenotype PMID:29289695
GO:0070994 Detection of oxidative stress Increases phenotype PMID:33508982; PMID:36000339
GO:0072593 Reactive oxygen species metabolic process Affects phenotype PMID:23922164; PMID:32663520; PMID:35164634
GO:0072672 Neutrophil extravasation Affects phenotype PMID:16508399; PMID:18382663; PMID:28003439
GO:0080009 Mrna methylation Affects phenotype PMID:35249202
GO:0090200 Positive regulation of release of cytochrome c from mitochondria Increases phenotype PMID:29289695
GO:0098609 Cell-cell adhesion Decreases phenotype PMID:27940100
GO:1900424 Regulation of defense response to bacterium Decreases phenotype PMID:28003439
GO:1901670 Negative regulation of superoxide dismutase activity Affects phenotype PMID:23922164
GO:1902110 Positive regulation of mitochondrial membrane permeability involved in apoptotic process Affects phenotype PMID:23922164
GO:1902552 Negative regulation of catalase activity Affects phenotype PMID:23922164
GO:1903409 Reactive oxygen species biosynthetic process Affects phenotype PMID:36027947
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:34553399
GO:2001235 Positive regulation of apoptotic signaling pathway Affects phenotype PMID:23922164

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.