Bromodichloromethane


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0002726 Positive regulation of t cell cytokine production Affects phenotype PMID:24211274
GO:0004129 Cytochrome-c oxidase activity Decreases phenotype PMID:25640707
GO:0006979 Response to oxidative stress Affects phenotype PMID:24211274
GO:0008177 Succinate dehydrogenase (ubiquinone) activity Decreases phenotype PMID:25640707
GO:0008284 Positive regulation of cell population proliferation Affects phenotype PMID:24001804
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:28321044
GO:0032729 Positive regulation of type ii interferon production Affects phenotype PMID:24211274
GO:0032731 Positive regulation of interleukin-1 beta production Affects phenotype PMID:24211274
GO:0032743 Positive regulation of interleukin-2 production Affects phenotype PMID:24211274
GO:0042246 Tissue regeneration Increases phenotype PMID:28321044
GO:0043065 Positive regulation of apoptotic process Affects phenotype PMID:24211274
GO:0044026 Dna hypermethylation Increases phenotype PMID:24001804
GO:0070994 Detection of oxidative stress Increases phenotype PMID:25640707

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.