Propyl p-hydroxybenzoate


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0000084 Mitotic s phase Affects phenotype PMID:33358972
GO:0000086 G2/m transition of mitotic cell cycle Affects phenotype PMID:33358972
GO:0001541 Ovarian follicle development Decreases phenotype PMID:22777679; PMID:28208728
GO:0001547 Antral ovarian follicle growth Affects phenotype PMID:31306770; PMID:32122340
GO:0001833 Inner cell mass cell proliferation Decreases phenotype PMID:40043921
GO:0001835 Blastocyst hatching Decreases phenotype PMID:40043921
GO:0006171 Camp biosynthetic process Increases phenotype PMID:26253279
GO:0006974 Cellular response to dna damage stimulus Increases phenotype PMID:31939680
GO:0007015 Actin filament organization Affects phenotype PMID:40043921
GO:0007566 Embryo implantation Decreases phenotype PMID:34101200
GO:0008283 Cell population proliferation Affects phenotype PMID:23744433; PMID:24481588; PMID:24481588; PMID:28855101; PMID:33358972
GO:0008284 Positive regulation of cell population proliferation Increases phenotype PMID:31939680
GO:0010424 Dna methylation on cytosine within a cg sequence Affects phenotype PMID:34523531
GO:0014049 Positive regulation of glutamate secretion Increases phenotype PMID:28174044
GO:0016049 Cell growth Decreases phenotype PMID:36277366
GO:0032125 Micronucleus organization Increases phenotype PMID:40043921
GO:0032148 Activation of protein kinase b activity Increases phenotype PMID:26253279
GO:0033148 Positive regulation of intracellular estrogen receptor signaling pathway Increases phenotype PMID:23567241; PMID:25449125; PMID:31939680
GO:0035936 Testosterone secretion Increases phenotype PMID:31306770
GO:0035938 Estradiol secretion Affects phenotype PMID:31306770; PMID:32122340
GO:0042310 Vasoconstriction Decreases phenotype PMID:2868457
GO:0042311 Vasodilation Increases phenotype PMID:2868457
GO:0044237 Cellular metabolic process Decreases phenotype PMID:36277366; PMID:37690743
GO:0044849 Estrous cycle Affects phenotype PMID:34365652
GO:0045333 Cellular respiration Affects phenotype PMID:33140513
GO:0045444 Fat cell differentiation Increases phenotype PMID:24155963
GO:0045992 Negative regulation of embryonic development Increases phenotype PMID:40043921
GO:0046884 Follicle-stimulating hormone secretion Affects phenotype PMID:28208728; PMID:32122340
GO:0048160 Primary follicle stage Affects phenotype PMID:22777679
GO:0051882 Mitochondrial depolarization Increases phenotype PMID:33140513
GO:0060207 Diestrus Increases phenotype PMID:28208728
GO:0060283 Negative regulation of oocyte development Increases phenotype PMID:34365652
GO:0070374 Positive regulation of erk1 and erk2 cascade Affects phenotype PMID:26253279
GO:1901216 Positive regulation of neuron death Increases phenotype PMID:28174044
GO:1903047 Mitotic cell cycle process Affects phenotype PMID:33358972
GO:1904078 Positive regulation of estrogen biosynthetic process Increases phenotype PMID:34365652
GO:1904457 Positive regulation of neuronal action potential Increases phenotype PMID:28174044
GO:2000863 Positive regulation of estrogen secretion Increases phenotype PMID:34101200; PMID:34365652
GO:2000872 Positive regulation of progesterone secretion Increases phenotype PMID:34101200
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.