Cyproconazole


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001755 Neural crest cell migration Decreases phenotype PMID:26238599
GO:0006641 Triglyceride metabolic process Affects phenotype PMID:32123961; PMID:32810590; PMID:34273450; PMID:34273450
GO:0006695 Cholesterol biosynthetic process Increases phenotype PMID:28454766
GO:0006754 Atp biosynthetic process Decreases phenotype PMID:29995386
GO:0007033 Vacuole organization Increases phenotype PMID:25182419; PMID:29038839
GO:0008283 Cell population proliferation Increases phenotype PMID:28058446
GO:0008610 Lipid biosynthetic process Increases phenotype PMID:28058446
GO:0019432 Triglyceride biosynthetic process Increases phenotype PMID:29995386
GO:0030520 Intracellular estrogen receptor signaling pathway Affects phenotype PMID:39365753
GO:0030521 Androgen receptor signaling pathway Affects phenotype PMID:39365753
GO:0045444 Fat cell differentiation Increases phenotype PMID:36951524
GO:0045795 Positive regulation of cell volume Increases phenotype PMID:29038839
GO:0047823 D-glutamyltransferase activity Increases phenotype PMID:29038839
GO:0048863 Stem cell differentiation Decreases phenotype PMID:32205227
GO:0050810 Regulation of steroid biosynthetic process Affects phenotype PMID:26781511
GO:0060992 Response to fungicide Affects phenotype PMID:23721867
GO:0140042 Lipid droplet formation Increases phenotype PMID:36951524
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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.