Acrylonitrile


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001541 Ovarian follicle development Affects phenotype PMID:35429547
GO:0001552 Ovarian follicle atresia Increases phenotype PMID:35429547
GO:0006304 Dna modification Increases phenotype PMID:26332274
GO:0006749 Glutathione metabolic process Affects phenotype PMID:39615733
GO:0006915 Apoptotic process Increases phenotype PMID:39615733
GO:0006954 Inflammatory response Increases phenotype PMID:35429547
GO:0007005 Mitochondrion organization Increases phenotype PMID:35429547; PMID:39615733
GO:0007137 Meiotic metaphase ii Decreases phenotype PMID:35429547
GO:0007612 Learning Affects phenotype PMID:39615733
GO:0007613 Memory Affects phenotype PMID:39615733
GO:0007628 Adult walking behavior Affects phenotype PMID:39615733
GO:0008283 Cell population proliferation Affects phenotype PMID:30236600
GO:0008285 Negative regulation of cell population proliferation Increases phenotype PMID:33132245
GO:0016042 Lipid catabolic process Increases phenotype PMID:12850505; PMID:33486070
GO:0016477 Cell migration Increases phenotype PMID:30236600
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:20981856
GO:0032418 Lysosome localization Affects phenotype PMID:35429547
GO:0042585 Germinal vesicle Affects phenotype PMID:35429547
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:33132245
GO:0044237 Cellular metabolic process Decreases phenotype PMID:33486070; PMID:39615733
GO:0046034 Atp metabolic process Affects phenotype PMID:39615733
GO:0050808 Synapse organization Affects phenotype PMID:39615733
GO:0051726 Regulation of cell cycle Increases phenotype PMID:33132245
GO:0051881 Regulation of mitochondrial membrane potential Affects phenotype PMID:39615733
GO:0051882 Mitochondrial depolarization Increases phenotype PMID:35429547; PMID:39615733
GO:0070813 Hydrogen sulfide metabolic process Affects phenotype PMID:33486070
GO:0070814 Hydrogen sulfide biosynthetic process Decreases phenotype PMID:33486070
GO:0070997 Neuron death Increases phenotype PMID:39615733
GO:0072593 Reactive oxygen species metabolic process Increases phenotype PMID:33486070; PMID:35429547; PMID:39615733
GO:1903409 Reactive oxygen species biosynthetic process Increases phenotype PMID:30236600
GO:1903428 Positive regulation of reactive oxygen species biosynthetic process Increases phenotype PMID:33132245
GO:1904700 Granulosa cell apoptotic process Increases phenotype PMID:35429547
GO:1905037 Autophagosome organization Increases phenotype PMID:39615733
DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.