| GO ID | GO name | Interaction type | Reference |
|---|---|---|---|
| GO:0006306 | Dna methylation | Affects phenotype | PMID:26339649 |
| GO:0006805 | Xenobiotic metabolic process | Increases phenotype | PMID:27818348 |
| GO:0006915 | Apoptotic process | Increases phenotype | PMID:8370474 |
| GO:0006974 | Cellular response to dna damage stimulus | Increases phenotype | PMID:25075717 |
| GO:0007049 | Cell cycle | Decreases phenotype | PMID:17984142 |
| GO:0007610 | Behavior | Decreases phenotype | PMID:36513211 |
| GO:0007613 | Memory | Decreases phenotype | PMID:36513211 |
| GO:0008203 | Cholesterol metabolic process | Affects phenotype | PMID:22421054 |
| GO:0008283 | Cell population proliferation | Affects phenotype | PMID:17984142 |
| GO:0010424 | Dna methylation on cytosine within a cg sequence | Affects phenotype | PMID:22851337; PMID:24169490; PMID:34009014 |
| GO:0010883 | Regulation of lipid storage | Affects phenotype | PMID:29617909 |
| GO:0031507 | Heterochromatin formation | Decreases phenotype | PMID:26339649 |
| GO:0035094 | Response to nicotine | Increases phenotype | PMID:27589885 |
| GO:0044030 | Regulation of dna methylation | Increases phenotype | PMID:22851337; PMID:37914285 |
| GO:0044237 | Cellular metabolic process | Affects phenotype | PMID:31678612 |
| GO:0045666 | Positive regulation of neuron differentiation | Increases phenotype | PMID:37914285 |
| GO:0045792 | Negative regulation of cell size | Increases phenotype | PMID:29617909; PMID:37914285 |
| GO:0050890 | Cognition | Decreases phenotype | PMID:36513211 |
| GO:1900017 | Positive regulation of cytokine production involved in inflammatory response | Increases phenotype | PMID:29617909 |
| GO:1902009 | Positive regulation of toxin transport | Decreases phenotype | PMID:29501574 |
We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.