Lead diacetate


Literature identifierStudy typeTest dosageEffective dosageEndocrine-mediated endpointsSystems-level perturbations
PMID:10091860IVR0.1 % 0.1 %Affects neuronal signalingNeurological endocrine-mediated perturbations
IVR0.5 % 0.5 %Affects neuronal signalingNeurological endocrine-mediated perturbations
IVR0.2 % 0.2 %Affects neuronal signalingNeurological endocrine-mediated perturbations
IVR1 % 1 %Affects neuronal signalingNeurological endocrine-mediated perturbations
PMID:21933222IVR0.05 mg/kg/day 0.05 mg/kg/dayOxidative stress in liverHepatic endocrine-mediated perturbations
IVR0.05 mg/kg/day 0.05 mg/kg/dayDecreased testosterone levelsReproductive endocrine-mediated perturbations
IVR0.05 mg/kg/day 0.05 mg/kg/dayDecreased cholesterol levelsMetabolic endocrine-mediated perturbations
IVR0.05 mg/kg/day 0.05 mg/kg/dayReduced sperm countsReproductive endocrine-mediated perturbations
IVR0.05 mg/kg/day 0.05 mg/kg/dayAffects steroidogenesisReproductive endocrine-mediated perturbations
IVR0.05 mg/kg/day 0.05 mg/kg/dayAffects sperm motilityReproductive endocrine-mediated perturbations
PMID:26558472IVR0.01 mg/L 0.01 mg/LAffects steroidogenesisReproductive endocrine-mediated perturbations
IVR0.1 mg/L 0.1 mg/LAffects steroidogenesisReproductive endocrine-mediated perturbations
PMID:33223335IVR1 - 25 mg/kg 1 - 25 mg/kgChanges in morphology of thyroid glandMetabolic endocrine-mediated perturbations
IVR1 - 25 mg/kg 1 - 25 mg/kgAlters hypothalamic-pituitary-thyroid axisMetabolic endocrine-mediated perturbations;Neurological endocrine-mediated perturbations
IVR1 - 25 mg/kg 1 - 25 mg/kgHypertrophy in thyroid glandMetabolic endocrine-mediated perturbations
IVR1 - 25 mg/kg 1 - 25 mg/kgInduce behavioral changesNeurological endocrine-mediated perturbations
IVR1 - 25 mg/kg 1 - 25 mg/kgIncrease in T4 levelsMetabolic endocrine-mediated perturbations
IVR1 - 25 mg/kg 1 - 25 mg/kgDecrease in T3 levelsMetabolic endocrine-mediated perturbations
PMID:37716076IVR5 mg/L 5 mg/LCauses inflammationImmunological endocrine-mediated perturbations
IVR5 mg/L 5 mg/LChanges in white blood cell (WBC) populationsImmunological endocrine-mediated perturbations
IVR5 mg/L 5 mg/LOxidative stress in testisReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LAffects steroidogenesisReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LDecreased testosterone levelsReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LAffects blood-testis barrier (BTB)Reproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LAbnormal sperm morphologyReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LAffects spermatogenesisReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LReduced sperm qualityReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LReduced sperm countsReproductive endocrine-mediated perturbations
IVR5 mg/L 5 mg/LAffects testicular morphologyReproductive endocrine-mediated perturbations
PMID:3931714IVR0.3 % 0.3 %Reduced sperm countsReproductive endocrine-mediated perturbations
IVR0.1 % 0.1 %Decreased FSH levelsReproductive endocrine-mediated perturbations
IVR0.3 % 0.3 %Increased prostate weightsReproductive endocrine-mediated perturbations
IVR0.1 % 0.1 %Reduced sperm countsReproductive endocrine-mediated perturbations
IVR0.3 % 0.3 %Decreased testosterone levelsReproductive endocrine-mediated perturbations
IVR0.1 % 0.1 %Decreased testosterone levelsReproductive endocrine-mediated perturbations
IVR0.1 % 0.1 %Increased prostate weightsReproductive endocrine-mediated perturbations
IVR0.3 % 0.3 %Decreased FSH levelsReproductive endocrine-mediated perturbations

DISCLAIMER

We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.