1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine


Curated chemical-phenotype interactions from CTD
GO IDGO nameInteraction typeReference
GO:0001694 Histamine biosynthetic process Increases phenotype PMID:30195017
GO:0001774 Microglial cell activation Increases phenotype PMID:22746536; PMID:24991814; PMID:25064079; PMID:37741537; PMID:38629493
GO:0001775 Cell activation Increases phenotype PMID:23562983
GO:0001780 Neutrophil homeostasis Decreases phenotype PMID:34808223
GO:0002260 Lymphocyte homeostasis Decreases phenotype PMID:34808223
GO:0002534 Cytokine production involved in inflammatory response Increases phenotype PMID:37741537
GO:0003990 Acetylcholinesterase activity Decreases phenotype PMID:30529163
GO:0004602 Glutathione peroxidase activity Decreases phenotype PMID:24768735
GO:0004784 Superoxide dismutase activity Affects phenotype PMID:24768735; PMID:38016618; PMID:38016618; PMID:38629493; PMID:38629493
GO:0006749 Glutathione metabolic process Increases phenotype PMID:23943375; PMID:27235848; PMID:36435477
GO:0006750 Glutathione biosynthetic process Decreases phenotype PMID:25053526
GO:0006805 Xenobiotic metabolic process Increases phenotype PMID:19594327
GO:0006809 Nitric oxide biosynthetic process Increases phenotype PMID:24991814
GO:0006915 Apoptotic process Increases phenotype PMID:23201480; PMID:24095822; PMID:24607817; PMID:24607817; PMID:29071302; PMID:32968928; PMID:33368846; PMID:35007524
GO:0006954 Inflammatory response Increases phenotype PMID:37741537; PMID:38629493
GO:0006997 Nucleus organization Decreases phenotype PMID:29071302
GO:0007033 Vacuole organization Increases phenotype PMID:33368846
GO:0007610 Behavior Decreases phenotype PMID:27235848
GO:0007626 Locomotory behavior Decreases phenotype PMID:25406165; PMID:32692843
GO:0008137 Nadh dehydrogenase (ubiquinone) activity Decreases phenotype PMID:10961998; PMID:23562983
GO:0008219 Cell death Increases phenotype PMID:16515773; PMID:18535749; PMID:18535749; PMID:24991814; PMID:22891246; PMID:22891246; PMID:25106480; PMID:25263579; PMID:30195017; PMID:24991814; PMID:25017139; PMID:25106480; PMID:25263579; PMID:27324791; PMID:28978077; PMID:30195017; PMID:8724674
GO:0008283 Cell population proliferation Decreases phenotype PMID:23538867; PMID:23933227; PMID:24995576; PMID:24995576; PMID:25064079; PMID:25064079; PMID:25248837; PMID:30529163
GO:0008344 Adult locomotory behavior Affects phenotype PMID:30195017
GO:0009407 Toxin catabolic process Decreases phenotype PMID:28807673
GO:0009448 Gamma-aminobutyric acid metabolic process Affects phenotype PMID:33368846
GO:0010942 Positive regulation of cell death Increases phenotype PMID:17555897; PMID:1933335; PMID:25693864
GO:0014067 Negative regulation of phosphatidylinositol 3-kinase signaling Affects phenotype PMID:23911879
GO:0014068 Positive regulation of phosphatidylinositol 3-kinase signaling Decreases phenotype PMID:28185818
GO:0015871 Choline transport Decreases phenotype PMID:11406107
GO:0016042 Lipid catabolic process Affects phenotype PMID:28092019; PMID:33368846
GO:0018158 Protein oxidation Increases phenotype PMID:27277809; PMID:27277809; PMID:28092019; PMID:28092019
GO:0019370 Leukotriene biosynthetic process Increases phenotype PMID:30195017
GO:0030263 Apoptotic chromosome condensation Increases phenotype PMID:29071302
GO:0031987 Locomotion involved in locomotory behavior Decreases phenotype PMID:27044752; PMID:27044752; PMID:28978077; PMID:28092019; PMID:28092019; PMID:33368846; PMID:36435477; PMID:38629493; PMID:28978077; PMID:33368846; PMID:36435477; PMID:38629493
GO:0032148 Activation of protein kinase b activity Decreases phenotype PMID:28185818
GO:0032273 Positive regulation of protein polymerization Increases phenotype PMID:34808223
GO:0032981 Mitochondrial respiratory chain complex i assembly Decreases phenotype PMID:29371327
GO:0033058 Directional locomotion Decreases phenotype PMID:24991814
GO:0033278 Cell proliferation in midbrain Decreases phenotype PMID:22511790
GO:0033687 Osteoblast proliferation Decreases phenotype PMID:30529163
GO:0034440 Lipid oxidation Increases phenotype PMID:23943375; PMID:36435477
GO:0034599 Cellular response to oxidative stress Affects phenotype PMID:24095822
GO:0035640 Exploration behavior Decreases phenotype PMID:33472166
GO:0035641 Locomotory exploration behavior Decreases phenotype PMID:37741537
GO:0035684 Helper t cell extravasation Increases phenotype PMID:31351185
GO:0036269 Swimming behavior Decreases phenotype PMID:30195017; PMID:33368846
GO:0040011 Locomotion Decreases phenotype PMID:24140863
GO:0042307 Positive regulation of protein import into nucleus Increases phenotype PMID:27566062
GO:0042416 Dopamine biosynthetic process Decreases phenotype PMID:10318960; PMID:23562983; PMID:27277809; PMID:28377118; PMID:30529163; PMID:23562983; PMID:27044752; PMID:27182044; PMID:27277809
GO:0042417 Dopamine metabolic process Increases phenotype PMID:23943375; PMID:23943375; PMID:30236862; PMID:24103762; PMID:27235848; PMID:28092019; PMID:28092019; PMID:28978077; PMID:32968928; PMID:38016618; PMID:28092019; PMID:33359019; PMID:28807673; PMID:28978077; PMID:28978077; PMID:33368846; PMID:32968928; PMID:33359019; PMID:33368846; PMID:38016618
GO:0042420 Dopamine catabolic process Decreases phenotype PMID:27182044
GO:0042428 Serotonin metabolic process Affects phenotype PMID:28978077
GO:0043065 Positive regulation of apoptotic process Increases phenotype PMID:15621629; PMID:23911879; PMID:25968939; PMID:29371327
GO:0043123 Positive regulation of i-kappab kinase/nf-kappab signaling Increases phenotype PMID:25406165
GO:0043525 Positive regulation of neuron apoptotic process Increases phenotype PMID:20133810; PMID:25406165; PMID:29371327
GO:0043552 Positive regulation of phosphatidylinositol 3-kinase activity Decreases phenotype PMID:28185818
GO:0043954 Cellular component maintenance Decreases phenotype PMID:28807673
GO:0044237 Cellular metabolic process Decreases phenotype PMID:35007524
GO:0045019 Negative regulation of nitric oxide biosynthetic process Increases phenotype PMID:31187328
GO:0045454 Cell redox homeostasis Affects phenotype PMID:27235848
GO:0045793 Positive regulation of cell size Increases phenotype PMID:28807673
GO:0046209 Nitric oxide metabolic process Affects phenotype PMID:35007524
GO:0046466 Membrane lipid catabolic process Increases phenotype PMID:19476553
GO:0048143 Astrocyte activation Increases phenotype PMID:38629493
GO:0050665 Hydrogen peroxide biosynthetic process Increases phenotype PMID:22891246
GO:0050729 Positive regulation of inflammatory response Increases phenotype PMID:32692843
GO:0050881 Musculoskeletal movement Affects phenotype PMID:31351185
GO:0051092 Positive regulation of nf-kappab transcription factor activity Increases phenotype PMID:32692843
GO:0051402 Neuron apoptotic process Increases phenotype PMID:32692843; PMID:36435477; PMID:37741537
GO:0051898 Negative regulation of protein kinase b signaling Affects phenotype PMID:23911879
GO:0055072 Iron ion homeostasis Affects phenotype PMID:36435477
GO:0060586 Multicellular organismal iron ion homeostasis Decreases phenotype PMID:25968939
GO:0061430 Bone trabecula morphogenesis Decreases phenotype PMID:30529163
GO:0061744 Motor behavior Decreases phenotype PMID:31119595; PMID:33472166
GO:0061890 Positive regulation of astrocyte activation Increases phenotype PMID:33035625
GO:0061900 Glial cell activation Increases phenotype PMID:32692843
GO:0070265 Necrotic cell death Increases phenotype PMID:33368846
GO:0070662 Mast cell proliferation Increases phenotype PMID:30195017
GO:0070994 Detection of oxidative stress Increases phenotype PMID:25053526; PMID:38016618; PMID:38016618; PMID:38629493; PMID:38629493
GO:0070997 Neuron death Increases phenotype PMID:10318960; PMID:22746536; PMID:27277809; PMID:28377118; PMID:33359019; PMID:28978077; PMID:28978077; PMID:29030221; PMID:37741537; PMID:38629493; PMID:29030221; PMID:33359019; PMID:33472166; PMID:37741537; PMID:37741537; PMID:38629493
GO:0072593 Reactive oxygen species metabolic process Affects phenotype PMID:33359019
GO:0090074 Negative regulation of protein homodimerization activity Increases phenotype PMID:31187328
GO:0090327 Negative regulation of locomotion involved in locomotory behavior Increases phenotype PMID:23943375; PMID:23943375; PMID:34808223; PMID:34808223; PMID:35104500
GO:0120056 Large intestinal transit Decreases phenotype PMID:31187328
GO:0120061 Negative regulation of gastric emptying Increases phenotype PMID:31187328
GO:1901082 Positive regulation of relaxation of smooth muscle Increases phenotype PMID:31187328
GO:1901216 Positive regulation of neuron death Increases phenotype PMID:19594327; PMID:23911879; PMID:23943375; PMID:23943375; PMID:28939911; PMID:31381935; PMID:35104500; PMID:25630971; PMID:28185818; PMID:28185818; PMID:31351185; PMID:28807673; PMID:28939911; PMID:31351185; PMID:31381935; PMID:34808223; PMID:35104500
GO:1901671 Positive regulation of superoxide dismutase activity Decreases phenotype PMID:25968939
GO:1902957 Negative regulation of mitochondrial electron transport, nadh to ubiquinone Increases phenotype PMID:29371327
GO:1903181 Positive regulation of dopamine biosynthetic process Decreases phenotype PMID:19594327
GO:1904124 Microglial cell migration Increases phenotype PMID:30195017
GO:1905319 Mesenchymal stem cell migration Increases phenotype PMID:30195017
GO:1905604 Negative regulation of blood-brain barrier permeability Increases phenotype PMID:31351185
GO:1990535 Neuron projection maintenance Decreases phenotype PMID:24103762; PMID:35104500
GO:2000821 Regulation of grooming behavior Decreases phenotype PMID:31119595

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We have built a comprehensive resource which compiles potential endocrine disrupting chemicals (EDCs) based on the observed adverse effects or endocrine-mediated endpoints in published experiments on humans or rodents to support basic research. We are not responsible for any errors or omissions in the published research articles or supporting literature on potential EDCs compiled in this resource. Users are advised to exercise their own judgement on the weight of evidence for potential EDCs compiled in this resource. Importantly, our sole goal to build this resource on potential EDCs is to enable future basic research towards better understanding of the systems-level perturbations upon chemical exposure rather than influencing regulatory advice on chemical use.